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A deficiency of the link protein Bral2 affects the size of the extracellular space in the thalamus of aged mice

The result's identifiers

  • Result code in IS VaVaI

    <a href="https://www.isvavai.cz/riv?ss=detail&h=RIV%2F00216208%3A11130%2F18%3A10375418" target="_blank" >RIV/00216208:11130/18:10375418 - isvavai.cz</a>

  • Alternative codes found

    RIV/68378041:_____/18:00481280

  • Result on the web

    <a href="https://doi.org/10.1002/jnr.24136" target="_blank" >https://doi.org/10.1002/jnr.24136</a>

  • DOI - Digital Object Identifier

    <a href="http://dx.doi.org/10.1002/jnr.24136" target="_blank" >10.1002/jnr.24136</a>

Alternative languages

  • Result language

    angličtina

  • Original language name

    A deficiency of the link protein Bral2 affects the size of the extracellular space in the thalamus of aged mice

  • Original language description

    Bral2 is a link protein stabilizing the binding between lecticans and hyaluronan in perineuronal nets and axonal coats (ACs) in specific brain regions. Using the real-time iontophoretic method and diffusion-weighted magnetic resonance, we determined the extracellular space (ECS) volume fraction (), tortuosity (), and apparent diffusion coefficient of water (ADC(W)) in the thalamic ventral posteromedial nucleus (VPM) and sensorimotor cortex of young adult (3-6 months) and aged (14-20 months) Bral2-deficient (Bral2(-/-)) mice and age-matched wild-type (wt) controls. The results were correlated with an analysis of extracellular matrix composition. In the cortex, no changes between wt and Bral2(-/-) were detected, either in the young or aged mice. In the VPM of aged but not in young Bral2(-/-) mice, we observed a significant decrease in and ADC(W) in comparison with age-matched controls. Bral2 deficiency led to a reduction of both aggrecan- and brevican-associated perineuronal nets and a complete disruption of brevican-based ACs in young as well as aged VPM. Our data suggest that aging is a critical point that reveals the effect of Bral2 deficiency on VPM diffusion. This effect is probably mediated through the enhanced age-related damage of neurons lacking protective ACs, or the exhausting of compensatory mechanisms maintaining unchanged diffusion parameters in young Bral2(-/-) animals. A decreased ECS volume in aged Bral2(-/-) mice may influence the diffusion of neuroactive substances, and thus extrasynaptic and also indirectly synaptic transmission in this important nucleus of the somatosensory pathway.

  • Czech name

  • Czech description

Classification

  • Type

    J<sub>imp</sub> - Article in a specialist periodical, which is included in the Web of Science database

  • CEP classification

  • OECD FORD branch

    30103 - Neurosciences (including psychophysiology)

Result continuities

  • Project

    <a href="/en/project/GA13-11867S" target="_blank" >GA13-11867S: The role of brain link proteins for formation and maintenance of perineuronal nets</a><br>

  • Continuities

    P - Projekt vyzkumu a vyvoje financovany z verejnych zdroju (s odkazem do CEP)

Others

  • Publication year

    2018

  • Confidentiality

    S - Úplné a pravdivé údaje o projektu nepodléhají ochraně podle zvláštních právních předpisů

Data specific for result type

  • Name of the periodical

    Journal of Neuroscience Research

  • ISSN

    0360-4012

  • e-ISSN

  • Volume of the periodical

    96

  • Issue of the periodical within the volume

    2

  • Country of publishing house

    US - UNITED STATES

  • Number of pages

    15

  • Pages from-to

    313-327

  • UT code for WoS article

    000417961000015

  • EID of the result in the Scopus database

    2-s2.0-85037718030