All

What are you looking for?

All
Projects
Results
Organizations

Quick search

  • Projects supported by TA ČR
  • Excellent projects
  • Projects with the highest public support
  • Current projects

Smart search

  • That is how I find a specific +word
  • That is how I leave the -word out of the results
  • “That is how I can find the whole phrase”

Molecular Targets for Cancer Therapy in the PI3K/AKT/mTOR Pathway

The result's identifiers

  • Result code in IS VaVaI

    <a href="https://www.isvavai.cz/riv?ss=detail&h=RIV%2F00216208%3A11140%2F14%3A10146044" target="_blank" >RIV/00216208:11140/14:10146044 - isvavai.cz</a>

  • Alternative codes found

    RIV/00669806:_____/14:10146044

  • Result on the web

    <a href="http://dx.doi.org/10.1016/j.pharmthera.2013.12.004" target="_blank" >http://dx.doi.org/10.1016/j.pharmthera.2013.12.004</a>

  • DOI - Digital Object Identifier

    <a href="http://dx.doi.org/10.1016/j.pharmthera.2013.12.004" target="_blank" >10.1016/j.pharmthera.2013.12.004</a>

Alternative languages

  • Result language

    angličtina

  • Original language name

    Molecular Targets for Cancer Therapy in the PI3K/AKT/mTOR Pathway

  • Original language description

    Aberrations in various cellular signaling pathways are instrumental in regulating cellular metabolism, tumor development, growth, proliferation, metastasis and cytoskeletal reorganization. The fundamental cellular signaling cascade involved in these processes, the phosphatidylinositol 3-kinase/protein kinase-B/mammalian target of rapamycin (PI3K/AKT/mTOR), closely related to the mitogen-activated protein kinase (MAPK) pathway, is a crucial and intensively explored intracellular signaling pathway in tumorigenesis. Various activating mutations in oncogenes together with the inactivation of tumor suppressor genes are found in diverse malignancies across almost all members of the pathway. Substantial progress in uncovering PI3K/AKT/mTOR alterations and their roles in tumorigenesis has enabled the development of novel targeted molecules with potential for developing efficacious anticancer treatment. Two approved anticancer drugs, everolimus and temsirolimus, exemplify targeted inhibition of

  • Czech name

  • Czech description

Classification

  • Type

    J<sub>x</sub> - Unclassified - Peer-reviewed scientific article (Jimp, Jsc and Jost)

  • CEP classification

    EB - Genetics and molecular biology

  • OECD FORD branch

Result continuities

  • Project

    <a href="/en/project/ED2.1.00%2F03.0076" target="_blank" >ED2.1.00/03.0076: Biomedical Centre of the Faculty of Medicine in Pilsen</a><br>

  • Continuities

    P - Projekt vyzkumu a vyvoje financovany z verejnych zdroju (s odkazem do CEP)

Others

  • Publication year

    2014

  • Confidentiality

    S - Úplné a pravdivé údaje o projektu nepodléhají ochraně podle zvláštních právních předpisů

Data specific for result type

  • Name of the periodical

    Pharmacology and Therapeutics

  • ISSN

    0163-7258

  • e-ISSN

  • Volume of the periodical

    142

  • Issue of the periodical within the volume

    2

  • Country of publishing house

    GB - UNITED KINGDOM

  • Number of pages

    12

  • Pages from-to

    164-175

  • UT code for WoS article

    000334135000003

  • EID of the result in the Scopus database