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Proteomic insights into chronic anthracycline cardiotoxicity

The result's identifiers

  • Result code in IS VaVaI

    <a href="https://www.isvavai.cz/riv?ss=detail&h=RIV%2F00216208%3A11150%2F11%3A10099465" target="_blank" >RIV/00216208:11150/11:10099465 - isvavai.cz</a>

  • Alternative codes found

    RIV/00216208:11160/11:10099465 RIV/60162694:G44__/11:00002570

  • Result on the web

    <a href="http://www.sciencedirect.com/science/article/pii/S0022282811000587" target="_blank" >http://www.sciencedirect.com/science/article/pii/S0022282811000587</a>

  • DOI - Digital Object Identifier

    <a href="http://dx.doi.org/10.1016/j.yjmcc.2011.01.018" target="_blank" >10.1016/j.yjmcc.2011.01.018</a>

Alternative languages

  • Result language

    angličtina

  • Original language name

    Proteomic insights into chronic anthracycline cardiotoxicity

  • Original language description

    In this study, global molecular changes associated with chronic anthracycline cardiotoxicity were analyzed for the first time using proteomic techniques. This approach identified 47 proteins significantly changed due to the treatment. Important changes were found in mitochondrial oxidative phosphorylation and energy channeling, intermediate filament desmin, myosin light chains, proteolytic systems, chaperon proteins and many others. Furthermore, basement membrane and extracellular matrix remodeling wasdocumented. The above described changes were corroborated with other molecular and biochemical methods. Hence, in this study the proteomic signature of chronic anthracycline cardiotoxicity was described and it enhances understanding to the molecular basis of this iatrogenic disease.

  • Czech name

  • Czech description

Classification

  • Type

    J<sub>x</sub> - Unclassified - Peer-reviewed scientific article (Jimp, Jsc and Jost)

  • CEP classification

    FR - Pharmacology and apothecary chemistry

  • OECD FORD branch

Result continuities

  • Project

    <a href="/en/project/GA305%2F09%2F0416" target="_blank" >GA305/09/0416: Myocardial damage induced by anticancer drugs and ischemia-reperfusion: new possibilities of pharmacological cardioprotection.</a><br>

  • Continuities

    P - Projekt vyzkumu a vyvoje financovany z verejnych zdroju (s odkazem do CEP)<br>Z - Vyzkumny zamer (s odkazem do CEZ)

Others

  • Publication year

    2011

  • Confidentiality

    S - Úplné a pravdivé údaje o projektu nepodléhají ochraně podle zvláštních právních předpisů

Data specific for result type

  • Name of the periodical

    Journal of Molecular and Cellular Cardiology

  • ISSN

    0022-2828

  • e-ISSN

  • Volume of the periodical

    50

  • Issue of the periodical within the volume

    5

  • Country of publishing house

    US - UNITED STATES

  • Number of pages

    14

  • Pages from-to

    849-862

  • UT code for WoS article

    000289748200012

  • EID of the result in the Scopus database