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Low zinc environment induces stress signaling, senescence and mixed cell death modalities in colon cancer cells

The result's identifiers

  • Result code in IS VaVaI

    <a href="https://www.isvavai.cz/riv?ss=detail&h=RIV%2F00216208%3A11150%2F15%3A10306938" target="_blank" >RIV/00216208:11150/15:10306938 - isvavai.cz</a>

  • Result on the web

    <a href="http://link.springer.com/article/10.1007/s10495-015-1182-5" target="_blank" >http://link.springer.com/article/10.1007/s10495-015-1182-5</a>

  • DOI - Digital Object Identifier

    <a href="http://dx.doi.org/10.1007/s10495-015-1182-5" target="_blank" >10.1007/s10495-015-1182-5</a>

Alternative languages

  • Result language

    angličtina

  • Original language name

    Low zinc environment induces stress signaling, senescence and mixed cell death modalities in colon cancer cells

  • Original language description

    Currently it is not clear what type of the final cellular response (i.e. cell death modality or senescence) is induced upon chronic intracellular zinc depletion in colon cancer cells. To address this question, isogenic colon cancer lines SW480 and SW620exposed to low zinc environment were studied over the period of 6 weeks. Low zinc environment reduced total as well as free intracellular zinc content in both cell lines. Decreased intracellular zinc content resulted in changes in cellular proliferation,cell cycle distribution and activation of stress signaling. In addition, colonocytes with low zinc content displayed increased levels of oxidative stress, changes in mitochondrial activity but in the absence of significant DNA damage. Towards the end oftreatment (4th-6th week), exposed cells started to change morphologically, and typical markers of senescence as well as cell death appeared. Of two examined colon cancer cell lines, SW480 cells proved to activate predominantly senescent

  • Czech name

  • Czech description

Classification

  • Type

    J<sub>x</sub> - Unclassified - Peer-reviewed scientific article (Jimp, Jsc and Jost)

  • CEP classification

    EB - Genetics and molecular biology

  • OECD FORD branch

Result continuities

  • Project

  • Continuities

    I - Institucionalni podpora na dlouhodoby koncepcni rozvoj vyzkumne organizace

Others

  • Publication year

    2015

  • Confidentiality

    S - Úplné a pravdivé údaje o projektu nepodléhají ochraně podle zvláštních právních předpisů

Data specific for result type

  • Name of the periodical

    Apoptosis : an international journal on programmed cell death

  • ISSN

    1360-8185

  • e-ISSN

  • Volume of the periodical

    20

  • Issue of the periodical within the volume

    12

  • Country of publishing house

    US - UNITED STATES

  • Number of pages

    15

  • Pages from-to

    1651-1665

  • UT code for WoS article

    000362881900009

  • EID of the result in the Scopus database

    2-s2.0-84943798316