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Flubendazole induces mitotic catastrophe and apoptosis in melanoma cells

The result's identifiers

  • Result code in IS VaVaI

    <a href="https://www.isvavai.cz/riv?ss=detail&h=RIV%2F00216208%3A11150%2F18%3A10377027" target="_blank" >RIV/00216208:11150/18:10377027 - isvavai.cz</a>

  • Alternative codes found

    RIV/00179906:_____/18:10377027

  • Result on the web

    <a href="https://doi.org/10.1016/j.tiv.2017.10.025" target="_blank" >https://doi.org/10.1016/j.tiv.2017.10.025</a>

  • DOI - Digital Object Identifier

    <a href="http://dx.doi.org/10.1016/j.tiv.2017.10.025" target="_blank" >10.1016/j.tiv.2017.10.025</a>

Alternative languages

  • Result language

    angličtina

  • Original language name

    Flubendazole induces mitotic catastrophe and apoptosis in melanoma cells

  • Original language description

    Flubendazole (FLU) is a widely used anthelmintic drug belonging to benzimidazole group. Recently, several studies have been published demonstrating its potential to inhibit growth of various tumor cells including those derived from colorectal cancer, breast cancer or leukemia via several mechanisms. In the present study we have investigated cytotoxic effects of FLU on malignant melanoma using A-375, BOWES and RPMI-7951 cell lines representing diverse melanoma molecular types. In all three cell lines, FLU inhibited cell growth and proliferation and disrupted microtubule structure and function which was accompanied by dramatic changes in cellular morphology. In addition, FLU-treated cells accumulated at the G2/M phase of cell cycle and displayed the features of mitotic catastrophe characterized by formation of giant cells with multiple nuclei, abnormal spindles and subsequent apoptotic demise. Although this endpoint was observed in all treated melanoma lines, our analyses showed different activated biochemical signaling in particular cells, thus suggesting a promising treatment potential of FLU in malignant melanoma warranting its further testing.

  • Czech name

  • Czech description

Classification

  • Type

    J<sub>imp</sub> - Article in a specialist periodical, which is included in the Web of Science database

  • CEP classification

  • OECD FORD branch

    30108 - Toxicology

Result continuities

  • Project

  • Continuities

    S - Specificky vyzkum na vysokych skolach<br>I - Institucionalni podpora na dlouhodoby koncepcni rozvoj vyzkumne organizace

Others

  • Publication year

    2018

  • Confidentiality

    S - Úplné a pravdivé údaje o projektu nepodléhají ochraně podle zvláštních právních předpisů

Data specific for result type

  • Name of the periodical

    Toxicology in Vitro

  • ISSN

    0887-2333

  • e-ISSN

  • Volume of the periodical

    46

  • Issue of the periodical within the volume

    February

  • Country of publishing house

    GB - UNITED KINGDOM

  • Number of pages

    10

  • Pages from-to

    313-322

  • UT code for WoS article

    000422811300036

  • EID of the result in the Scopus database

    2-s2.0-85033399921