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The influence of ochratoxin A on DNA adduct formation by the carcinogen aristolochic acid in rats

The result's identifiers

  • Result code in IS VaVaI

    <a href="https://www.isvavai.cz/riv?ss=detail&h=RIV%2F00216208%3A11310%2F15%3A10313493" target="_blank" >RIV/00216208:11310/15:10313493 - isvavai.cz</a>

  • Result on the web

    <a href="http://link.springer.com/article/10.1007/s00204-014-1360-1#/page-1" target="_blank" >http://link.springer.com/article/10.1007/s00204-014-1360-1#/page-1</a>

  • DOI - Digital Object Identifier

    <a href="http://dx.doi.org/10.1007/s00204-014-1360-1" target="_blank" >10.1007/s00204-014-1360-1</a>

Alternative languages

  • Result language

    angličtina

  • Original language name

    The influence of ochratoxin A on DNA adduct formation by the carcinogen aristolochic acid in rats

  • Original language description

    Exposure to the plant nephrotoxin and carcinogen aristolochic acid (AA) leads to the development of AA nephropathy, Balkan endemic nephropathy (BEN) and upper urothelial carcinoma (UUC) in humans. Beside AA, exposure to ochratoxin A (OTA) was linked to BEN. Although OTA was rejected as a factor for BEN/UUC, there is still no information whether the development of AAinduced BEN/UUC is influenced by OTA exposure. Therefore, we studied the influence of OTA on the genotoxicity of AA (AA-DNA adduct formation) in vivo. AA-DNA adducts were formed in liver and kidney of rats treated with AA or AA combined with OTA, but no OTA-related DNA adducts were detectable in rats treated with OTA alone or OTA combined with AA. Compared to rats treated with AA alone, AA-DNA adduct levels were 5.4- and 1.6-fold higher in liver and kidney, respectively, of rats treated with AA combined with OTA. Although AA and OTA induced NAD(P)H:quinone oxidoreductase (NQO1) activating AA to DNA adducts, their combined tr

  • Czech name

  • Czech description

Classification

  • Type

    J<sub>x</sub> - Unclassified - Peer-reviewed scientific article (Jimp, Jsc and Jost)

  • CEP classification

    CE - Biochemistry

  • OECD FORD branch

Result continuities

  • Project

    <a href="/en/project/GBP303%2F12%2FG163" target="_blank" >GBP303/12/G163: Centre of drug-dietary supplements interactions and nutrigenetics</a><br>

  • Continuities

    S - Specificky vyzkum na vysokych skolach

Others

  • Publication year

    2015

  • Confidentiality

    S - Úplné a pravdivé údaje o projektu nepodléhají ochraně podle zvláštních právních předpisů

Data specific for result type

  • Name of the periodical

    Archives of Toxicology

  • ISSN

    0340-5761

  • e-ISSN

  • Volume of the periodical

    89

  • Issue of the periodical within the volume

    11

  • Country of publishing house

    DE - GERMANY

  • Number of pages

    18

  • Pages from-to

    2141-2158

  • UT code for WoS article

    000365417800020

  • EID of the result in the Scopus database

    2-s2.0-84947573720