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Signals trigger state-specific transcriptional programs to support diversity and homeostasis in immune cells

The result's identifiers

  • Result code in IS VaVaI

    <a href="https://www.isvavai.cz/riv?ss=detail&h=RIV%2F00216224%3A14740%2F19%3A00113240" target="_blank" >RIV/00216224:14740/19:00113240 - isvavai.cz</a>

  • Result on the web

    <a href="https://stke.sciencemag.org/content/12/581/eaao5820" target="_blank" >https://stke.sciencemag.org/content/12/581/eaao5820</a>

  • DOI - Digital Object Identifier

    <a href="http://dx.doi.org/10.1126/scisignal.aao5820" target="_blank" >10.1126/scisignal.aao5820</a>

Alternative languages

  • Result language

    angličtina

  • Original language name

    Signals trigger state-specific transcriptional programs to support diversity and homeostasis in immune cells

  • Original language description

    Macrophages play key roles in the immune systems of humans and other mammals. Here, we performed single-cell analyses of the mRNAs and proteins of human macrophages to compare their responses to the signaling molecules lipopolysaccharide (LPS), a component of Gram-negative bacteria, and palmitate (PAL), a free fatty acid. We found that, although both molecules signal through the cell surface protein Toll-like receptor 4 (TLR4), they stimulated the expression of different genes, resulting in specific pro- and anti-inflammatory cellular states for each signal. The effects of the glucocorticoid receptor, which antagonizes LPS signaling, and cyclic AMP-dependent transcription factor 3, which inhibits PAL-induced inflammation, on inflammatory response seemed largely determined by digital on-off events. Furthermore, the quantification of transcriptional variance and signaling entropy enabled the identification of cell state-specific deregulated molecular pathways. These data suggest that the preservation of signaling in distinct cells might confer diversity on macrophage populations essential to maintaining major cellular functions.

  • Czech name

  • Czech description

Classification

  • Type

    J<sub>imp</sub> - Article in a specialist periodical, which is included in the Web of Science database

  • CEP classification

  • OECD FORD branch

    10608 - Biochemistry and molecular biology

Result continuities

  • Project

  • Continuities

    I - Institucionalni podpora na dlouhodoby koncepcni rozvoj vyzkumne organizace

Others

  • Publication year

    2019

  • Confidentiality

    S - Úplné a pravdivé údaje o projektu nepodléhají ochraně podle zvláštních právních předpisů

Data specific for result type

  • Name of the periodical

    SCIENCE SIGNALING

  • ISSN

    1945-0877

  • e-ISSN

  • Volume of the periodical

    12

  • Issue of the periodical within the volume

    581

  • Country of publishing house

    US - UNITED STATES

  • Number of pages

    16

  • Pages from-to

    1-16

  • UT code for WoS article

    000467963000001

  • EID of the result in the Scopus database

    2-s2.0-85066279813