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The Biological Effects of Complete Gasoline Engine Emissions Exposure in a 3D Human Airway Model (MucilAir(TM)) and in Human Bronchial Epithelial Cells (BEAS-2B)

The result's identifiers

  • Result code in IS VaVaI

    <a href="https://www.isvavai.cz/riv?ss=detail&h=RIV%2F60460709%3A41310%2F19%3A81925" target="_blank" >RIV/60460709:41310/19:81925 - isvavai.cz</a>

  • Alternative codes found

    RIV/68378041:_____/19:00518531 RIV/68407700:21220/19:00335128 RIV/68407700:21230/19:00335128 RIV/00216208:11310/19:10409254

  • Result on the web

    <a href="https://www.mdpi.com/1422-0067/20/22/5710" target="_blank" >https://www.mdpi.com/1422-0067/20/22/5710</a>

  • DOI - Digital Object Identifier

    <a href="http://dx.doi.org/10.3390/ijms20225710" target="_blank" >10.3390/ijms20225710</a>

Alternative languages

  • Result language

    angličtina

  • Original language name

    The Biological Effects of Complete Gasoline Engine Emissions Exposure in a 3D Human Airway Model (MucilAir(TM)) and in Human Bronchial Epithelial Cells (BEAS-2B)

  • Original language description

    The biological effects induced by complete engine emissions in a 3D model of the human airway (MucilAir(TM)) and in human bronchial epithelial cells (BEAS-2B) grown at the air-liquid interface were compared. The cells were exposed for one or five days to emissions generated by a Euro 5 direct injection spark ignition engine. The general condition of the cells was assessed by the measurement of transepithelial electrical resistance and mucin production. The cytotoxic effects were evaluated by adenylate kinase (AK) and lactate dehydrogenase (LDH) activity. Phosphorylation of histone H2AX was used to detect double-stranded DNA breaks. The expression of the selected 370 relevant genes was analyzed using next-generation sequencing. The exposure had minimal effects on integrity and AK leakage in both cell models. LDH activity and mucin production in BEAS-2B cells significantly increased after longer exposures DNA breaks were also detected. The exposure affected CYP1A1 and HSPA5 expression in MucilAir(TM).

  • Czech name

  • Czech description

Classification

  • Type

    J<sub>imp</sub> - Article in a specialist periodical, which is included in the Web of Science database

  • CEP classification

  • OECD FORD branch

    10608 - Biochemistry and molecular biology

Result continuities

  • Project

  • Continuities

    S - Specificky vyzkum na vysokych skolach

Others

  • Publication year

    2019

  • Confidentiality

    S - Úplné a pravdivé údaje o projektu nepodléhají ochraně podle zvláštních právních předpisů

Data specific for result type

  • Name of the periodical

    International Journal of Molecular Sciences

  • ISSN

    1422-0067

  • e-ISSN

    1422-0067

  • Volume of the periodical

    2019

  • Issue of the periodical within the volume

    20

  • Country of publishing house

    CH - SWITZERLAND

  • Number of pages

    22

  • Pages from-to

    1-22

  • UT code for WoS article

    000502786800192

  • EID of the result in the Scopus database