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DDI2 protease controls embryonic development and inflammation via TCF11/NRF1

The result's identifiers

  • Result code in IS VaVaI

    <a href="https://www.isvavai.cz/riv?ss=detail&h=RIV%2F61388963%3A_____%2F24%3A00598945" target="_blank" >RIV/61388963:_____/24:00598945 - isvavai.cz</a>

  • Alternative codes found

    RIV/68378050:_____/24:00598945 RIV/00216208:11310/24:10484773 RIV/00216208:11110/24:10484773

  • Result on the web

    <a href="https://doi.org/10.1016/j.isci.2024.110893" target="_blank" >https://doi.org/10.1016/j.isci.2024.110893</a>

  • DOI - Digital Object Identifier

    <a href="http://dx.doi.org/10.1016/j.isci.2024.110893" target="_blank" >10.1016/j.isci.2024.110893</a>

Alternative languages

  • Result language

    angličtina

  • Original language name

    DDI2 protease controls embryonic development and inflammation via TCF11/NRF1

  • Original language description

    DDI2 is an aspartic protease that cleaves polyubiquitinated substrates. Upon proteotoxic stress, DDI2 activates the transcription factor TCF11/NRF1 (NFE2L1), crucial for maintaining proteostasis in mammalian cells, enabling the expression of rescue factors, including proteasome subunits. Here, we describe the consequences of DDI2 ablation in vivo and in cells. DDI2 knock-out (KO) in mice caused embryonic lethality at E12.5 with severe developmental failure. Molecular characterization of embryos showed insufficient proteasome expression with proteotoxic stress, accumulation of high molecular weight ubiquitin conjugates and induction of the unfolded protein response (UPR) and cell death pathways. In DDI2 surrogate KO cells, proteotoxic stress activated the integrated stress response (ISR) and induced a type I interferon (IFN) signature and IFN-induced proliferative signaling, possibly ensuring survival. These results indicate an important role for DDI2 in the cell-tissue proteostasis network and in maintaining a balanced immune response.

  • Czech name

  • Czech description

Classification

  • Type

    J<sub>imp</sub> - Article in a specialist periodical, which is included in the Web of Science database

  • CEP classification

  • OECD FORD branch

    10608 - Biochemistry and molecular biology

Result continuities

  • Project

    Result was created during the realization of more than one project. More information in the Projects tab.

  • Continuities

    I - Institucionalni podpora na dlouhodoby koncepcni rozvoj vyzkumne organizace

Others

  • Publication year

    2024

  • Confidentiality

    S - Úplné a pravdivé údaje o projektu nepodléhají ochraně podle zvláštních právních předpisů

Data specific for result type

  • Name of the periodical

    iScience

  • ISSN

    2589-0042

  • e-ISSN

    2589-0042

  • Volume of the periodical

    27

  • Issue of the periodical within the volume

    10

  • Country of publishing house

    US - UNITED STATES

  • Number of pages

    26

  • Pages from-to

    110893

  • UT code for WoS article

    001318582000001

  • EID of the result in the Scopus database

    2-s2.0-85207346406