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Heterogenous mutation spectrum and deregulated cellular pathways in aberrant plasma cells underline molecular pathology of light-chain amyloidosis

The result's identifiers

  • Result code in IS VaVaI

    <a href="https://www.isvavai.cz/riv?ss=detail&h=RIV%2F61988987%3A17110%2F21%3AA22028RK" target="_blank" >RIV/61988987:17110/21:A22028RK - isvavai.cz</a>

  • Alternative codes found

    RIV/00669806:_____/21:10427706 RIV/00843989:_____/21:E0108826 RIV/00216208:11140/21:10427706 RIV/65269705:_____/21:00074366 and 3 more

  • Result on the web

    <a href="https://haematologica.org/article/view/9744" target="_blank" >https://haematologica.org/article/view/9744</a>

  • DOI - Digital Object Identifier

    <a href="http://dx.doi.org/10.3324/haematol.2019.239756" target="_blank" >10.3324/haematol.2019.239756</a>

Alternative languages

  • Result language

    angličtina

  • Original language name

    Heterogenous mutation spectrum and deregulated cellular pathways in aberrant plasma cells underline molecular pathology of light-chain amyloidosis

  • Original language description

    Light-chain (AL) amyloidosis (ALA) is a rare but fatal monoclonal gammopathy (MG) causing organ and tissue damage resulting from the deposition of misfolded immunoglobulin free light chains in the form of amyloid fibrils.1 In some cases, ALA coexists with multiple myeloma (MM) (ALA+MM), which is the second most common blood cancer and is caused by the proliferation of clonal plasma cells (PC).2 Due to insufficient knowledge of ALA and ALA+MM biology, therapeutic options have mirrored treatment regimens of MM, which focus on the elimination of clonal PC.3,4 We investigated the mutation and gene expression profiles in clonal aberrant PC (aPC) in order to better understand ALA and ALA+MM etiology and to clarify the molecular differences between individual MG diagnoses.

  • Czech name

  • Czech description

Classification

  • Type

    J<sub>imp</sub> - Article in a specialist periodical, which is included in the Web of Science database

  • CEP classification

  • OECD FORD branch

    30205 - Hematology

Result continuities

  • Project

    <a href="/en/project/NV15-29667A" target="_blank" >NV15-29667A: Aberrant plasma cells clonal diversification in immunoglobulin light chain amyloidosis</a><br>

  • Continuities

    P - Projekt vyzkumu a vyvoje financovany z verejnych zdroju (s odkazem do CEP)

Others

  • Publication year

    2021

  • Confidentiality

    S - Úplné a pravdivé údaje o projektu nepodléhají ochraně podle zvláštních právních předpisů

Data specific for result type

  • Name of the periodical

    HAEMATOL-HEMATOL J

  • ISSN

    0390-6078

  • e-ISSN

    1592-8721

  • Volume of the periodical

    106

  • Issue of the periodical within the volume

    2

  • Country of publishing house

    IT - ITALY

  • Number of pages

    4

  • Pages from-to

    601-604

  • UT code for WoS article

    000615767000010

  • EID of the result in the Scopus database

    2-s2.0-85100634389