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Deoxynivalenol (Vomitoxin)-induced anorexia is induced by the release of intestinal hormones in mice

The result's identifiers

  • Result code in IS VaVaI

    <a href="https://www.isvavai.cz/riv?ss=detail&h=RIV%2F62690094%3A18470%2F21%3A50018239" target="_blank" >RIV/62690094:18470/21:50018239 - isvavai.cz</a>

  • Alternative codes found

    RIV/00179906:_____/21:10431120

  • Result on the web

    <a href="https://www.mdpi.com/2072-6651/13/8/512" target="_blank" >https://www.mdpi.com/2072-6651/13/8/512</a>

  • DOI - Digital Object Identifier

    <a href="http://dx.doi.org/10.3390/toxins13080512" target="_blank" >10.3390/toxins13080512</a>

Alternative languages

  • Result language

    angličtina

  • Original language name

    Deoxynivalenol (Vomitoxin)-induced anorexia is induced by the release of intestinal hormones in mice

  • Original language description

    Deoxynivalenol (DON), also known as vomitoxin, is a mycotoxin that can cause antifeeding and vomiting in animals. However, the mechanism of DON inducing anorexia is complicated. Studies have shown that intestinal hormones play a significant part in the anorexia caused by DON. We adopted the “modeling of acute antifeeding in mice” as the basic experimental model, and used two methods of gavage and intraperitoneal injection to explore the effect of intestinal hormones on the antifeedant response induced by DON in mice. We found that 1 and 2.5 mg/kg·bw of DON can acutely induce anorexia and increase the plasma intestinal hormones CCK, PYY, GIP, and GLP-1 in mice within 3 h. Direct injection of exogenous intestinal hormones CCK, PYY, GIP, and GLP-1 can trigger anorexia behavior in mice. Furthermore, the PYY receptor antagonist JNJ-31020028, GLP-1 receptor antagonist Exendin(9-39), CCK receptor antagonist Proglumide, GIP receptor antagonist GIP(3-30)NH2 attenuated both intestinal hormone and DON-induced anorectic responses. These results indicate that intestinal hormones play a critical role in the anorexia response induced by DON. © 2021 by the authors. Licensee MDPI, Basel, Switzerland.

  • Czech name

  • Czech description

Classification

  • Type

    J<sub>imp</sub> - Article in a specialist periodical, which is included in the Web of Science database

  • CEP classification

  • OECD FORD branch

    30108 - Toxicology

Result continuities

  • Project

  • Continuities

    I - Institucionalni podpora na dlouhodoby koncepcni rozvoj vyzkumne organizace

Others

  • Publication year

    2021

  • Confidentiality

    S - Úplné a pravdivé údaje o projektu nepodléhají ochraně podle zvláštních právních předpisů

Data specific for result type

  • Name of the periodical

    Toxins

  • ISSN

    2072-6651

  • e-ISSN

  • Volume of the periodical

    13

  • Issue of the periodical within the volume

    8

  • Country of publishing house

    CH - SWITZERLAND

  • Number of pages

    14

  • Pages from-to

    "Article number 512"

  • UT code for WoS article

    000689956500001

  • EID of the result in the Scopus database

    2-s2.0-85111469392