Reactive oxygen species, cell growth, cell cycle progression and vascular remodeling in hypertension

Result code in IS VaVaI

<a href="https://www.isvavai.cz/riv?ss=detail&h=RIV%2F67985823%3A_____%2F07%3A00331655" target="_blank" >RIV/67985823:_____/07:00331655 - isvavai.cz</a>

Result on the web

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DOI - Digital Object Identifier

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Result language

angličtina

Original language name

Reactive oxygen species, cell growth, cell cycle progression and vascular remodeling in hypertension

Original language description

Reactive oxygen species (ROS) are important intracellular signaling molecules. During increased oxidative stress and associated oxidative damage ROS are mediators of vascular injury in hypertension. Oxidative stress causes vascular injury by reducing nitric oxide bioavailability, altering endothelial function and vascular contraction/dilation, promoting vascular smooth muscle cell proliferation and hypertrophy, and increasing extracellular matrix deposition and inflammation. The present review focuses on the regulatory role of ROS on cell growth and cell cycle progression

Czech name

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Czech description

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Type

J_x - Unclassified - Peer-reviewed scientific article (Jimp, Jsc and Jost)

CEP classification

ED - Physiology

OECD FORD branch

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Project

<a href="/en/project/1M0510" target="_blank" >1M0510: Centre for cardiovascular research</a><br>

Continuities

Z - Vyzkumny zamer (s odkazem do CEZ)

Publication year

2007

Confidentiality

S - Úplné a pravdivé údaje o projektu nepodléhají ochraně podle zvláštních právních předpisů

Name of the periodical

Future Cardiology

ISSN

1479-6678

e-ISSN

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Volume of the periodical

3

Issue of the periodical within the volume

1

Country of publishing house

GB - UNITED KINGDOM

Number of pages

11

Pages from-to

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UT code for WoS article

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EID of the result in the Scopus database

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