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CKS1 Germ Line Exclusion Is Essential for the Transition from Meiosis to Early Embryonic Development

The result's identifiers

  • Result code in IS VaVaI

    <a href="https://www.isvavai.cz/riv?ss=detail&h=RIV%2F67985904%3A_____%2F19%3A00512107" target="_blank" >RIV/67985904:_____/19:00512107 - isvavai.cz</a>

  • Alternative codes found

    RIV/00216208:11310/19:10403763

  • Result on the web

    <a href="https://mcb.asm.org/content/39/13/e00590-18" target="_blank" >https://mcb.asm.org/content/39/13/e00590-18</a>

  • DOI - Digital Object Identifier

    <a href="http://dx.doi.org/10.1128/MCB.00590-18" target="_blank" >10.1128/MCB.00590-18</a>

Alternative languages

  • Result language

    angličtina

  • Original language name

    CKS1 Germ Line Exclusion Is Essential for the Transition from Meiosis to Early Embryonic Development

  • Original language description

    Cell division cycle (cdc) kinase subunit (CKS) proteins bind cyclin-dependent kinases (CDKs) and play important roles in cell division control and development, though their precise molecular functions are not fully understood. Mammals express two closely related paralogs called CKS1 and CKS2, but only CKS2 is expressed in the germ line, indicating that it is solely responsible for regulating CDK functions in meiosis. Using cks2(-/-) knockout mice, we show that CKS2 is a crucial regulator of maturation-promoting factor (MPF, CDK1-cyclin A/B) activity in meiosis. cks2(-/-) oocytes display reduced and delayed MPF activity during meiotic progression, leading to defects in germinal vesicle breakdown (GVBD), anaphase-promoting complex/cyclosome (APC/C) activation, and meiotic spindle assembly. cks2(-/-) germ cells express significantly reduced levels of the MPF components CDK1 and cyclins A1/B1. Additionally, injection of MPF plus CKS2, but not MPF alone, restored normal GVBD in cks2(-/- )oocytes, demonstrating that GVBD is driven by a CKS2-dependent function of MPF. Moreover, we generated cks2(cks1/cks1)( )knock-in mice and found that CKS1 can compensate for CKS2 in meiosis in vivo, but homozygous embryos arrested development at the 2- to 5-cell stage. Collectively, our results show that CKS2 is a crucial regulator of MPF functions in meiosis and that its paralog, CKS1, must be excluded from the germ line for proper embryonic development.

  • Czech name

  • Czech description

Classification

  • Type

    J<sub>imp</sub> - Article in a specialist periodical, which is included in the Web of Science database

  • CEP classification

  • OECD FORD branch

    10601 - Cell biology

Result continuities

  • Project

    Result was created during the realization of more than one project. More information in the Projects tab.

  • Continuities

    P - Projekt vyzkumu a vyvoje financovany z verejnych zdroju (s odkazem do CEP)

Others

  • Publication year

    2019

  • Confidentiality

    S - Úplné a pravdivé údaje o projektu nepodléhají ochraně podle zvláštních právních předpisů

Data specific for result type

  • Name of the periodical

    Molecular and Cellular Biology

  • ISSN

    0270-7306

  • e-ISSN

  • Volume of the periodical

    39

  • Issue of the periodical within the volume

    13

  • Country of publishing house

    US - UNITED STATES

  • Number of pages

    18

  • Pages from-to

    UNSP e00590-18

  • UT code for WoS article

    000471243000005

  • EID of the result in the Scopus database

    2-s2.0-85068118528