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Modulation of death receptor-mediated apoptosis in differentiating human myeloid leukemia HL-60 cells.

The result's identifiers

  • Result code in IS VaVaI

    <a href="https://www.isvavai.cz/riv?ss=detail&h=RIV%2F68081707%3A_____%2F01%3A17013051" target="_blank" >RIV/68081707:_____/01:17013051 - isvavai.cz</a>

  • Alternative codes found

    RIV/00209805:_____/01:00004750

  • Result on the web

  • DOI - Digital Object Identifier

Alternative languages

  • Result language

    angličtina

  • Original language name

    Modulation of death receptor-mediated apoptosis in differentiating human myeloid leukemia HL-60 cells.

  • Original language description

    Cellular differentiation can play an important role in cell sensitivity to apoptotic stimuli. In the present study, effects of dimethyl sulfoxide (DMSO)- and all-trans retinoic acid (ATRA)-induced differentiation on tumor necrosis factor-alpha (TNF-alpha)- and anti-Fas-induced apoptosis in human myeloid leukemia cell line were studied. It was shown that DMSO potentiates, while ATRA blocks TNF-alpha and anti-Fas-induced apoptosis, in a time dependent manner

  • Czech name

  • Czech description

Classification

  • Type

    J<sub>x</sub> - Unclassified - Peer-reviewed scientific article (Jimp, Jsc and Jost)

  • CEP classification

    BO - Biophysics

  • OECD FORD branch

Result continuities

  • Project

    Result was created during the realization of more than one project. More information in the Projects tab.

  • Continuities

    Z - Vyzkumny zamer (s odkazem do CEZ)

Others

  • Publication year

    2001

  • Confidentiality

    S - Úplné a pravdivé údaje o projektu nepodléhají ochraně podle zvláštních právních předpisů

Data specific for result type

  • Name of the periodical

    Journal of Leukocyte Biology

  • ISSN

    0741-5400

  • e-ISSN

  • Volume of the periodical

    69

  • Issue of the periodical within the volume

    5

  • Country of publishing house

    US - UNITED STATES

  • Number of pages

    9

  • Pages from-to

    794-802

  • UT code for WoS article

  • EID of the result in the Scopus database