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Multipotency and therapeutic potential of NG2 cells.

The result's identifiers

  • Result code in IS VaVaI

    <a href="https://www.isvavai.cz/riv?ss=detail&h=RIV%2F68378041%3A_____%2F17%3A00476751" target="_blank" >RIV/68378041:_____/17:00476751 - isvavai.cz</a>

  • Result on the web

    <a href="http://dx.doi.org/10.1016/j.bcp.2017.05.008" target="_blank" >http://dx.doi.org/10.1016/j.bcp.2017.05.008</a>

  • DOI - Digital Object Identifier

    <a href="http://dx.doi.org/10.1016/j.bcp.2017.05.008" target="_blank" >10.1016/j.bcp.2017.05.008</a>

Alternative languages

  • Result language

    angličtina

  • Original language name

    Multipotency and therapeutic potential of NG2 cells.

  • Original language description

    NG2 cells represent one of the most proliferative glial cell populations in the intact mammalian central nervous system (CNS). They are well-known for their ability to renew themselves or to generate new oligodendrocytes during development as well as in adulthood, therefore also being termed oligodendrocyte progenitor cells. Following CNS injuries, such as demyelination, trauma or ischemia, the proliferative capacity of NG2 cells rapidly increases and moreover, their differentiation potential broadens, as documented by numerous reports also describing their differentiation into astrocytes or even neurons. Here, we summarize the current knowledge about NG2 cells proliferation, their fate plasticity during embryogenesis as well as in postnatal CNS under physiological and pathological conditions, with the main emphasis on the role of various signaling molecules, growth factors, hormones or even neurotransmitters on the fate potential of NG2 cells. (c) 2017 Elsevier Inc. All rights reserved.

  • Czech name

  • Czech description

Classification

  • Type

    J<sub>imp</sub> - Article in a specialist periodical, which is included in the Web of Science database

  • CEP classification

  • OECD FORD branch

    30103 - Neurosciences (including psychophysiology)

Result continuities

  • Project

    <a href="/en/project/GA17-04034S" target="_blank" >GA17-04034S: Principal signaling pathways regulating NG2 glia proliferation and differentiation following brain injuries</a><br>

  • Continuities

    P - Projekt vyzkumu a vyvoje financovany z verejnych zdroju (s odkazem do CEP)

Others

  • Publication year

    2017

  • Confidentiality

    S - Úplné a pravdivé údaje o projektu nepodléhají ochraně podle zvláštních právních předpisů

Data specific for result type

  • Name of the periodical

    Biochemical Pharmacology

  • ISSN

    0006-2952

  • e-ISSN

  • Volume of the periodical

    141

  • Issue of the periodical within the volume

    SI

  • Country of publishing house

    US - UNITED STATES

  • Number of pages

    13

  • Pages from-to

    42-55

  • UT code for WoS article

    000411656600004

  • EID of the result in the Scopus database

    2-s2.0-85019637465