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Mutations in GFAP Alter Early Lineage Commitment of Organoids

The result's identifiers

  • Result code in IS VaVaI

    <a href="https://www.isvavai.cz/riv?ss=detail&h=RIV%2F68378041%3A_____%2F25%3A00638093" target="_blank" >RIV/68378041:_____/25:00638093 - isvavai.cz</a>

  • Alternative codes found

    RIV/86652036:_____/25:00638351 RIV/00216208:11310/25:10515586

  • Result on the web

    <a href="https://onlinelibrary.wiley.com/doi/10.1002/glia.70049" target="_blank" >https://onlinelibrary.wiley.com/doi/10.1002/glia.70049</a>

  • DOI - Digital Object Identifier

    <a href="http://dx.doi.org/10.1002/glia.70049" target="_blank" >10.1002/glia.70049</a>

Alternative languages

  • Result language

    angličtina

  • Original language name

    Mutations in GFAP Alter Early Lineage Commitment of Organoids

  • Original language description

    Glial fibrillary acidic protein (GFAP) is a type-3 intermediate filament protein mainly expressed in astrocytes in the central nervous system. Mutations in GFAP cause Alexander disease (AxD), a rare and fatal neurological disorder. How exactly mutant GFAP eventually leads to white and gray matter deterioration in AxD remains unknown. GFAP is known to be expressed also in neural precursor cells in the developing brain. Here, we used AxD patient-derived induced pluripotent stem cells (iPSCs) to explore the impact of mutant GFAP during neurodifferentiation. Our results show that GFAP is already expressed in iPSCs. Moreover, we have found that mutations in GFAP can severely affect neural organoid development through altering lineage commitment in embryoid bodies. Together, these results support the notion that GFAP plays a role as an early modulator of neurodevelopment.

  • Czech name

  • Czech description

Classification

  • Type

    J<sub>imp</sub> - Article in a specialist periodical, which is included in the Web of Science database

  • CEP classification

  • OECD FORD branch

    30103 - Neurosciences (including psychophysiology)

Result continuities

  • Project

    Result was created during the realization of more than one project. More information in the Projects tab.

  • Continuities

    I - Institucionalni podpora na dlouhodoby koncepcni rozvoj vyzkumne organizace

Others

  • Publication year

    2025

  • Confidentiality

    S - Úplné a pravdivé údaje o projektu nepodléhají ochraně podle zvláštních právních předpisů

Data specific for result type

  • Name of the periodical

    Glia

  • ISSN

    0894-1491

  • e-ISSN

    1098-1136

  • Volume of the periodical

    73

  • Issue of the periodical within the volume

    11

  • Country of publishing house

    US - UNITED STATES

  • Number of pages

    22

  • Pages from-to

    2167-2188

  • UT code for WoS article

    001540289700001

  • EID of the result in the Scopus database

    2-s2.0-105011937143