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FBXO38 is dispensable for PD-1 regulation

The result's identifiers

  • Result code in IS VaVaI

    <a href="https://www.isvavai.cz/riv?ss=detail&h=RIV%2F68378050%3A_____%2F24%3A00599981" target="_blank" >RIV/68378050:_____/24:00599981 - isvavai.cz</a>

  • Alternative codes found

    RIV/00216208:11310/24:10487496

  • Result on the web

    <a href="https://www.embopress.org/doi/full/10.1038/s44319-024-00220-8" target="_blank" >https://www.embopress.org/doi/full/10.1038/s44319-024-00220-8</a>

  • DOI - Digital Object Identifier

    <a href="http://dx.doi.org/10.1038/s44319-024-00220-8" target="_blank" >10.1038/s44319-024-00220-8</a>

Alternative languages

  • Result language

    angličtina

  • Original language name

    FBXO38 is dispensable for PD-1 regulation

  • Original language description

    SKP1-CUL1-F-box protein (SCF) ubiquitin ligases are versatile protein complexes that mediate the ubiquitination of protein substrates. The direct substrate recognition relies on a large family of F-box-domain-containing subunits. One of these substrate receptors is FBXO38, which is encoded by a gene found mutated in families with early-onset distal motor neuronopathy. SCFFBXO38 ubiquitin ligase controls the stability of ZXDB, a nuclear factor associated with the centromeric chromatin protein CENP-B. Loss of FBXO38 in mice results in growth retardation and defects in spermatogenesis characterized by deregulation of the Sertoli cell transcription program and compromised centromere integrity. Moreover, it was reported that SCFFBXO38 mediates the degradation of PD-1, a key immune-checkpoint inhibitor in T cells. Here, we have re-addressed the link between SCFFBXO38 and PD-1 proteolysis. Our data do not support the notion that SCFFBXO38 directly or indirectly controls the abundance and stability of PD-1 in T cells.

  • Czech name

  • Czech description

Classification

  • Type

    J<sub>imp</sub> - Article in a specialist periodical, which is included in the Web of Science database

  • CEP classification

  • OECD FORD branch

    30102 - Immunology

Result continuities

  • Project

    Result was created during the realization of more than one project. More information in the Projects tab.

  • Continuities

    P - Projekt vyzkumu a vyvoje financovany z verejnych zdroju (s odkazem do CEP)

Others

  • Publication year

    2024

  • Confidentiality

    S - Úplné a pravdivé údaje o projektu nepodléhají ochraně podle zvláštních právních předpisů

Data specific for result type

  • Name of the periodical

    Embo Reports

  • ISSN

    1469-221X

  • e-ISSN

    1469-3178

  • Volume of the periodical

    25

  • Issue of the periodical within the volume

    10

  • Country of publishing house

    US - UNITED STATES

  • Number of pages

    20

  • Pages from-to

    4206-4225

  • UT code for WoS article

    001312686300001

  • EID of the result in the Scopus database

    2-s2.0-85203847699