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Jag1 insufficiency alters liver fibrosis via T cell and hepatocyte differentiation defects

The result's identifiers

  • Result code in IS VaVaI

    <a href="https://www.isvavai.cz/riv?ss=detail&h=RIV%2F68378050%3A_____%2F24%3A00602614" target="_blank" >RIV/68378050:_____/24:00602614 - isvavai.cz</a>

  • Alternative codes found

    RIV/00216208:11310/24:10489744

  • Result on the web

    <a href="https://www.embopress.org/doi/full/10.1038/s44321-024-00145-8" target="_blank" >https://www.embopress.org/doi/full/10.1038/s44321-024-00145-8</a>

  • DOI - Digital Object Identifier

    <a href="http://dx.doi.org/10.1038/s44321-024-00145-8" target="_blank" >10.1038/s44321-024-00145-8</a>

Alternative languages

  • Result language

    angličtina

  • Original language name

    Jag1 insufficiency alters liver fibrosis via T cell and hepatocyte differentiation defects

  • Original language description

    Fibrosis contributes to tissue repair, but excessive fibrosis disrupts organ function. Alagille syndrome (ALGS, caused by mutations in JAGGED1) results in liver disease and characteristic fibrosis. Here, we show that Jag1Ndr/Ndr mice, a model for ALGS, recapitulate ALGS-like fibrosis. Single-cell RNA-seq and multi-color flow cytometry of the liver revealed immature hepatocytes and paradoxically low intrahepatic T cell infiltration despite cholestasis in Jag1Ndr/Ndr mice. Thymic and splenic regulatory T cells (Tregs) were enriched and Jag1Ndr/Ndr lymphocyte immune and fibrotic capacity was tested with adoptive transfer into Rag1-/- mice, challenged with dextran sulfate sodium (DSS) or bile duct ligation (BDL). Transplanted Jag1Ndr/Ndr lymphocytes were less inflammatory with fewer activated T cells than Jag1+/+ lymphocytes in response to DSS. Cholestasis induced by BDL in Rag1-/- mice with Jag1Ndr/Ndr lymphocytes resulted in periportal Treg accumulation and three-fold less periportal fibrosis than in Rag1-/- mice with Jag1+/+ lymphocytes. Finally, the Jag1Ndr/Ndr hepatocyte expression profile and Treg overrepresentation were corroborated in patients' liver samples. Jag1-dependent hepatic and immune defects thus interact to determine the fibrotic process in ALGS.

  • Czech name

  • Czech description

Classification

  • Type

    J<sub>imp</sub> - Article in a specialist periodical, which is included in the Web of Science database

  • CEP classification

  • OECD FORD branch

    10601 - Cell biology

Result continuities

  • Project

    Result was created during the realization of more than one project. More information in the Projects tab.

  • Continuities

    I - Institucionalni podpora na dlouhodoby koncepcni rozvoj vyzkumne organizace

Others

  • Publication year

    2024

  • Confidentiality

    S - Úplné a pravdivé údaje o projektu nepodléhají ochraně podle zvláštních právních předpisů

Data specific for result type

  • Name of the periodical

    EMBO Molecular Medicine

  • ISSN

    1757-4676

  • e-ISSN

    1757-4684

  • Volume of the periodical

    16

  • Issue of the periodical within the volume

    11

  • Country of publishing house

    US - UNITED STATES

  • Number of pages

    30

  • Pages from-to

    2946-2975

  • UT code for WoS article

    001326988900001

  • EID of the result in the Scopus database

    2-s2.0-85205438394