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Integrative miRNOMe profiling reveals the miR-195-5p-CHEK1 axis and its impact on luminal breast cancer outcomes

The result's identifiers

  • Result code in IS VaVaI

    <a href="https://www.isvavai.cz/riv?ss=detail&h=RIV%2F75010330%3A_____%2F25%3A00015037" target="_blank" >RIV/75010330:_____/25:00015037 - isvavai.cz</a>

  • Alternative codes found

    RIV/00216208:11120/25:43928597 RIV/00216208:11130/25:10497379 RIV/00216208:11140/25:10497379 RIV/70883521:28150/25:63594818 RIV/00064203:_____/25:10497379

  • Result on the web

    <a href="https://febs.onlinelibrary.wiley.com/doi/10.1002/1878-0261.70077" target="_blank" >https://febs.onlinelibrary.wiley.com/doi/10.1002/1878-0261.70077</a>

  • DOI - Digital Object Identifier

    <a href="http://dx.doi.org/10.1002/1878-0261.70077" target="_blank" >10.1002/1878-0261.70077</a>

Alternative languages

  • Result language

    angličtina

  • Original language name

    Integrative miRNOMe profiling reveals the miR-195-5p-CHEK1 axis and its impact on luminal breast cancer outcomes

  • Original language description

    The luminal subtype (estrogen receptor-positive, ER+) is the most common and the most heterogeneous type of breast carcinoma (BC) in women. During our study, we determined expression levels of all microRNAs (miRNome) in 101 ER+ BC samples and identified 25 miRNAs being associated with proliferative markers. Using comprehensive in silico analyses we prioritized CHEK1, CDC25A, and CCNE1 as candidate genes affecting the proliferation of ER+ BC, with two microRNAs from the miR-497 similar to 195 cluster identified as their potential regulators. In a cohort of 217 patients, we found a significant association between high expression of CHEK1 and shorter relapse-free survival (RFS) in luminal BC patients treated with adjuvant chemotherapy, especially in patients with luminal A subtype. In patients treated with neoadjuvant therapy, the opposite role for RFS was observed for hsa-miR-195-5p. Subsequently, we confirmed the potency of hsa-miR-195-5p to inhibit the expression of CHEK1 in vitro. Moreover, the specific Chk1 inhibitor rabusertib (LY2603618) significantly enhanced the efficacy of doxorubicin in both ER+ and ER- cell lines. In summary, we have identified the association of a specific miRNA profile with highly proliferative luminal BCs and demonstrated the ability of hsa-miR-195-5p to inhibit CHEK1 expression in BC in vitro, underlining the importance of CHEK1 expression and its inhibition for prognosis and treatment of patients with luminal BCs.

  • Czech name

  • Czech description

Classification

  • Type

    J<sub>imp</sub> - Article in a specialist periodical, which is included in the Web of Science database

  • CEP classification

  • OECD FORD branch

    30204 - Oncology

Result continuities

  • Project

    Result was created during the realization of more than one project. More information in the Projects tab.

  • Continuities

    P - Projekt vyzkumu a vyvoje financovany z verejnych zdroju (s odkazem do CEP)

Others

  • Publication year

    2025

  • Confidentiality

    S - Úplné a pravdivé údaje o projektu nepodléhají ochraně podle zvláštních právních předpisů

Data specific for result type

  • Name of the periodical

    Molecular Oncology

  • ISSN

    1574-7891

  • e-ISSN

    1878-0261

  • Volume of the periodical

    19

  • Issue of the periodical within the volume

    11

  • Country of publishing house

    US - UNITED STATES

  • Number of pages

    18

  • Pages from-to

    3409-3426

  • UT code for WoS article

    001513100900001

  • EID of the result in the Scopus database

    2-s2.0-105008753747