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The role of DISP3 protein in lipid metabolism

Project goals

In the body the brain is the most cholesterol rich organ. Despite this, remarkably little is known about the mechanisms in the brain that regulate cholesterol homeostasis. By screening for T3 regulated genes we have identified Disp3, a sterol-sensing domain containing protein. DISP3 localizes within the endoplasmic reticulum and was further found to co-localize with cholesterol. Ectopic expression of DISP3 in fibroblasts resulted in elevated cholesterol levels combined with an altered cholesterol distribution. Currently, it is unknown what is responsible for the alteration of cholesterol in cells with modulated DISP3 expression and whether this protein interacts directly with cholesterol or other lipids. During the proposed project we will attempt to further characterize the function of DISP3 with regards to the regulation of cholesterol homeostasis and to study the potential physical interaction between DISP3 and cholesterol by in vitro and in vivo methods. In addition, we will establish the importance of the sterol-sensing domain for modulating cellular cholesterol distribution.

Keywords

sterol-sensingdomaincholesterollipidsmetabolism

Public support

  • Provider

    Czech Science Foundation

  • Programme

    Standard projects

  • Call for proposals

    Standardní projekty 15 (SGA02012GA-ST)

  • Main participants

    Ústav molekulární genetiky AV ČR, v. v. i.

  • Contest type

    VS - Public tender

  • Contract ID

    P301-12-1478

Alternative language

  • Project name in Czech

    Úloha proteinu DISP3 v lipidovém metabolismu

  • Annotation in Czech

    Přestože mozek obsahuje značné množství cholesterolu, je poměrně málo známo o mechanismech jeho regulace v tomto orgánu. Při hledání nových genů, které jsou regulovány thyroidním hormonem jsme identifikovali Disp3, nového člena SSD (sterol-sensing domain) proteinové rodiny. Vnitrobuněčně se DISP3 nachází v oblasti endoplazmatického retikula, kde kolokalizuje s cholesterolem. Ektopická exprese DISP3 ve fibroblastech vede k relokalizaci a k akumulaci celkového cholesterolu v buňce. V současné době není známo co způsobuje změnu cholesterolu v buňkách s modulovanou expresí DISP3 a zda DISP3 interaguje přímo s cholesterolem či s jinými lipidy. V předkládaném projektu chceme dále charakterizovat funkci DISP3 v homeostáze buněčného cholesterolu a dále pak studovat přímou interakci mezi DISP3 proteinem a cholesterolem, popřípadě dalšími steroly, a to pomocí in vitro a in vivo metod. Dále bychom rádi objasnili úlohu SSD-domény, popřípadě dalších domén, v této interakci.

Scientific branches

  • R&D category

    ZV - Basic research

  • CEP classification - main branch

    FB - Endocrinology, diabetology, metabolism, nutrition

  • CEP - secondary branch

    CE - Biochemistry

  • CEP - another secondary branch

    EB - Genetics and molecular biology

  • 10603 - Genetics and heredity (medical genetics to be 3)
    10604 - Reproductive biology (medical aspects to be 3)
    10605 - Developmental biology
    10608 - Biochemistry and molecular biology
    10609 - Biochemical research methods
    30101 - Human genetics
    30202 - Endocrinology and metabolism (including diabetes, hormones)

Completed project evaluation

  • Provider evaluation

    U - Uspěl podle zadání (s publikovanými či patentovanými výsledky atd.)

  • Project results evaluation

    The main goal of the project was to characterize function of DISP3, relevant in lipid metabolism and cholesterol levels. Investigators fulfilled the aims of the application and obtained results would enable further research, particularly in the field of oncogenetics. The project, which contributed to the education of the students, has resulted in three publications in sound journals (e.g. NPG).

Solution timeline

  • Realization period - beginning

    Jan 1, 2012

  • Realization period - end

    Apr 25, 2017

  • Project status

    U - Finished project

  • Latest support payment

    Mar 26, 2015

Data delivery to CEP

  • Confidentiality

    S - Úplné a pravdivé údaje o projektu nepodléhají ochraně podle zvláštních právních předpisů

  • Data delivery code

    CEP18-GA0-GA-U/02:1

  • Data delivery date

    May 4, 2018

Finance

  • Total approved costs

    7,448 thou. CZK

  • Public financial support

    7,448 thou. CZK

  • Other public sources

    0 thou. CZK

  • Non public and foreign sources

    0 thou. CZK

Basic information

Recognised costs

7 448 CZK thou.

Public support

7 448 CZK thou.

100%


Provider

Czech Science Foundation

CEP

FB - Endocrinology, diabetology, metabolism, nutrition

Solution period

01. 01. 2012 - 25. 04. 2017