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Statins and Inflammation

The result's identifiers

  • Result code in IS VaVaI

    <a href="https://www.isvavai.cz/riv?ss=detail&h=RIV%2F00023001%3A_____%2F21%3A00081787" target="_blank" >RIV/00023001:_____/21:00081787 - isvavai.cz</a>

  • Alternative codes found

    RIV/00216208:11110/21:10434704 RIV/00064165:_____/21:10434704

  • Result on the web

    <a href="https://link.springer.com/content/pdf/10.1007/s11883-021-00977-6.pdf" target="_blank" >https://link.springer.com/content/pdf/10.1007/s11883-021-00977-6.pdf</a>

  • DOI - Digital Object Identifier

    <a href="http://dx.doi.org/10.1007/s11883-021-00977-6" target="_blank" >10.1007/s11883-021-00977-6</a>

Alternative languages

  • Result language

    angličtina

  • Original language name

    Statins and Inflammation

  • Original language description

    Purpose of Review Chronic inflammation has been recognized as one of the most important pathophysiological mechanisms&apos; initiation and progression of atherosclerosis. Statins belong to most successful therapeutic agents in the prevention and treatment of atherothrombotic vascular disease. Their non-lipid related effects including suppression of inflammation have been repeatedly proven in both experimental and clinical settings. Recent Findings Recently, the importance of inflammation in the process of atherosclerosis has been confirmed by interventions targeting inflammation selectively. Clinical trial with selective inhibitor of a principal inflammatory mediator interleukin 1-beta - canakinumab - confirmed the notion of direct vasculoprotective effects of primarily targeting inflammation. This has increased interest in the non-lipid, pleiotropic and, particularly, anti-inflammatory effects of statins. Summary Anti-inflammatory effects of statins have been proven both experimentally and in clinical settings beyond any doubt. They comprise a direct positive effect on not only many cell types and pathways that are lipid independent but, also, some that are mediated by lipid modification. Undoubtedly, suppression of inflammatory response by statins contributes to their generally positive action in atherosclerosis and represents an important part of the vasculo- and atheroprotective effect of this drug class.

  • Czech name

  • Czech description

Classification

  • Type

    J<sub>imp</sub> - Article in a specialist periodical, which is included in the Web of Science database

  • CEP classification

  • OECD FORD branch

    30201 - Cardiac and Cardiovascular systems

Result continuities

  • Project

    <a href="/en/project/NU21-01-00146" target="_blank" >NU21-01-00146: Analysis of free DNA and miRNA as markers of muscle damage in statin-treated patients</a><br>

  • Continuities

    P - Projekt vyzkumu a vyvoje financovany z verejnych zdroju (s odkazem do CEP)

Others

  • Publication year

    2021

  • Confidentiality

    S - Úplné a pravdivé údaje o projektu nepodléhají ochraně podle zvláštních právních předpisů

Data specific for result type

  • Name of the periodical

    Current atherosclerosis reports

  • ISSN

    1523-3804

  • e-ISSN

  • Volume of the periodical

    23

  • Issue of the periodical within the volume

    12

  • Country of publishing house

    NL - THE KINGDOM OF THE NETHERLANDS

  • Number of pages

    8

  • Pages from-to

    "art. no. 80"

  • UT code for WoS article

    000724753700001

  • EID of the result in the Scopus database

    2-s2.0-85120355548