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Early onset of apc/c activity renders sac inefficient in mouse embryos

The result's identifiers

  • Result code in IS VaVaI

    <a href="https://www.isvavai.cz/riv?ss=detail&h=RIV%2F00027162%3A_____%2F24%3AN0000021" target="_blank" >RIV/00027162:_____/24:N0000021 - isvavai.cz</a>

  • Alternative codes found

    RIV/67985904:_____/24:00584848 RIV/00216224:14310/24:00135724

  • Result on the web

    <a href="https://www.frontiersin.org/articles/10.3389/fcell.2024.1355979/abstract" target="_blank" >https://www.frontiersin.org/articles/10.3389/fcell.2024.1355979/abstract</a>

  • DOI - Digital Object Identifier

    <a href="http://dx.doi.org/10.3389/fcell.2024.1355979" target="_blank" >10.3389/fcell.2024.1355979</a>

Alternative languages

  • Result language

    angličtina

  • Original language name

    Early onset of apc/c activity renders sac inefficient in mouse embryos

  • Original language description

    Control mechanisms of spindle assembly and chromosome segregaDon are vital for prevenDng aneuploidy during cell division. The mammalian germ cells and embryos are prone to chromosome segregaDon errors and resulDng aneuploidy is a major cause of terminaDon of development or severe developmental disorders. Here we focused on early mouse embryos, and using combinaDon of methods involving microinjecDon, immunodetecDon and confocal live cell imaging, we concentrated on the Spindle Assembly Checkpoint (SAC) and Anaphase PromoDng Complex/Cyclosome (APC/C). These are two important mechanisms cooperaDng during mitosis to ensure accurate chromosome segregaDon, and assessed their acDvity during the first two mitoses a[er ferDlizaDon. Our results showed, that in zygotes and 2-cell embryos, the SAC core protein Mad1 shows very low levels on kinetochores in comparison to oocytes and its interacDon with chromosomes is restricted to a short Dme interval a[er nuclear membrane disassembly (NEBD). Exposure of 2-cell embryos to low levels of spindle poison does not prevent anaphase, despite the spindle damage induced by the drug. And lastly, the APC/C is acDvated coincidentally with NEBD, before the spindle assembly compleDon. This early onset of APC/C acDvity, together with precocious relocalizaDon of Mad1 from chromosomes, prevents proper surveillance of spindle assembly by SAC. The results contribute to the understanding of the origin of aneuploidy in early embryos.

  • Czech name

  • Czech description

Classification

  • Type

    J<sub>imp</sub> - Article in a specialist periodical, which is included in the Web of Science database

  • CEP classification

  • OECD FORD branch

    40203 - Husbandry

Result continuities

  • Project

    <a href="/en/project/GA20-25850S" target="_blank" >GA20-25850S: Control of anaphase entry during early development</a><br>

  • Continuities

    P - Projekt vyzkumu a vyvoje financovany z verejnych zdroju (s odkazem do CEP)<br>I - Institucionalni podpora na dlouhodoby koncepcni rozvoj vyzkumne organizace

Others

  • Publication year

    2024

  • Confidentiality

    S - Úplné a pravdivé údaje o projektu nepodléhají ochraně podle zvláštních právních předpisů

Data specific for result type

  • Name of the periodical

    Frontiers in Cell and Developmental Biology

  • ISSN

    2296-634X

  • e-ISSN

    2296-634X

  • Volume of the periodical

    12

  • Issue of the periodical within the volume

    2024

  • Country of publishing house

    CH - SWITZERLAND

  • Number of pages

    13

  • Pages from-to

  • UT code for WoS article

    001190896200001

  • EID of the result in the Scopus database

    2-s2.0-85188590577