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Anti-Genotoxic Potential of Bilirubin In Vivo: Damage to DNA in Hyperbilirubinemic Human and Animal Models

The result's identifiers

  • Result code in IS VaVaI

    <a href="https://www.isvavai.cz/riv?ss=detail&h=RIV%2F00216208%3A11110%2F13%3A10192311" target="_blank" >RIV/00216208:11110/13:10192311 - isvavai.cz</a>

  • Result on the web

    <a href="http://dx.doi.org/10.1158/1940-6207.CAPR-13-0125" target="_blank" >http://dx.doi.org/10.1158/1940-6207.CAPR-13-0125</a>

  • DOI - Digital Object Identifier

    <a href="http://dx.doi.org/10.1158/1940-6207.CAPR-13-0125" target="_blank" >10.1158/1940-6207.CAPR-13-0125</a>

Alternative languages

  • Result language

    angličtina

  • Original language name

    Anti-Genotoxic Potential of Bilirubin In Vivo: Damage to DNA in Hyperbilirubinemic Human and Animal Models

  • Original language description

    The bile pigment bilirubin is a known antioxidant and is associated with protection from cancer and cardiovascular disease (CVD) when present in too strong concentrations. Unconjugated bilirubin (UCB) might also possess anti-genotoxic potential by preventing oxidative damage to DNA. Moderately elevated bilirubin levels are found in individuals with Gilbert syndrome and more severe in the hyperbilirubinemic Gunn rat model. This study was therefore aimed to assess the levels of oxidative damage to DNA inGilbert syndrome subjects and Gunn rats compared tomatched controls. Seventy-six individuals (age- and sex-matched) were allocated into Gilbert syndrome (UCB }= 17.1 mu mol/L; n - 38) or control groups (UCB < 17.1 mu mol/L; n = 38). In addition, 40 Gunnrats were used to support the results of the human trial. Single-cell gel electrophoresis (SCGE) assay measuring standard conditions (strand breaks, apurinic/apyrimidinic sites) and formamidopyrimidine glycosylase (FPG)-sensitive sites wa

  • Czech name

  • Czech description

Classification

  • Type

    J<sub>x</sub> - Unclassified - Peer-reviewed scientific article (Jimp, Jsc and Jost)

  • CEP classification

    CE - Biochemistry

  • OECD FORD branch

Result continuities

  • Project

  • Continuities

    N - Vyzkumna aktivita podporovana z neverejnych zdroju

Others

  • Publication year

    2013

  • Confidentiality

    S - Úplné a pravdivé údaje o projektu nepodléhají ochraně podle zvláštních právních předpisů

Data specific for result type

  • Name of the periodical

    Cancer Prevention Research

  • ISSN

    1940-6207

  • e-ISSN

  • Volume of the periodical

    6

  • Issue of the periodical within the volume

    10

  • Country of publishing house

    US - UNITED STATES

  • Number of pages

    8

  • Pages from-to

    1056-1063

  • UT code for WoS article

    000325272400007

  • EID of the result in the Scopus database