Role of FAT/CD36 in novel PKC isoform activation in heart of spontaneously hypertensive rats

Result code in IS VaVaI
<a href="https://www.isvavai.cz/riv?ss=detail&h=RIV%2F00216208%3A11310%2F11%3A10104518" target="_blank" >RIV/00216208:11310/11:10104518 - isvavai.cz</a>
Alternative codes found
RIV/67985823:_____/11:00365731 RIV/00023001:_____/11:00002503
Result on the web
<a href="http://dx.doi.org/10.1007/s11010-011-0886-2" target="_blank" >http://dx.doi.org/10.1007/s11010-011-0886-2</a>
DOI - Digital Object Identifier
<a href="http://dx.doi.org/10.1007/s11010-011-0886-2" target="_blank" >10.1007/s11010-011-0886-2</a>

Result language
angličtina
Original language name
Role of FAT/CD36 in novel PKC isoform activation in heart of spontaneously hypertensive rats
Original language description
Searching for a possible role of novel protein kinase C (nPKC) in heart with disrupted energy balance, we compared the insulin-resistant spontaneously hypertensive rats (SHR), which carry a nonfunctional variant of the fatty acid transporter FAT/CD36, with the less insulin-resistant congenic strain SHR-4 that is genetically identical except for a segment on chromosome 4 including a wild-type gene for a functional FAT/CD36. Our results demonstrate that reduced insulin resistance in SHR-4 rats is associated with the insulin signaling pathway involving nPKCs.
Czech name
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Czech description
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Type
J<sub>x</sub> - Unclassified - Peer-reviewed scientific article (Jimp, Jsc and Jost)
CEP classification
FB - Endocrinology, diabetology, metabolism, nutrition
OECD FORD branch
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Project
Result was created during the realization of more than one project. More information in the Projects tab.
Continuities
P - Projekt vyzkumu a vyvoje financovany z verejnych zdroju (s odkazem do CEP)<br>Z - Vyzkumny zamer (s odkazem do CEZ)<br>S - Specificky vyzkum na vysokych skolach

Publication year
2011
Confidentiality
S - Úplné a pravdivé údaje o projektu nepodléhají ochraně podle zvláštních právních předpisů

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