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Involvement of calcium signaling in different types of cell death in cancer

The result's identifiers

  • Result code in IS VaVaI

    <a href="https://www.isvavai.cz/riv?ss=detail&h=RIV%2F00216224%3A14110%2F22%3A00129730" target="_blank" >RIV/00216224:14110/22:00129730 - isvavai.cz</a>

  • Result on the web

    <a href="http://www.elis.sk/index.php?page=shop.product_details&flypage=flypage.tpl&product_id=7567&category_id=180&option=com_virtuemart&vmcchk=1&Itemid=1" target="_blank" >http://www.elis.sk/index.php?page=shop.product_details&flypage=flypage.tpl&product_id=7567&category_id=180&option=com_virtuemart&vmcchk=1&Itemid=1</a>

  • DOI - Digital Object Identifier

    <a href="http://dx.doi.org/10.4149/neo_2022_220127N121" target="_blank" >10.4149/neo_2022_220127N121</a>

Alternative languages

  • Result language

    angličtina

  • Original language name

    Involvement of calcium signaling in different types of cell death in cancer

  • Original language description

    Programmed cell death is a basic feature of chemotherapeutic (and also radiotherapeutic) intervention. Induction of cell death in tumor cells aims to kill preferentially the tumor cells, with minimal impact on the normal cells. Although apoptosis is the most obvious type of cell death induced by chemotherapeutics, several other types can also be activated, especially in conditions, where cells are resistant to apoptosis induction. Calcium signaling was shown to play an indisputable role in the activation of different types of cell death. Local increase of the calcium in time and precise place of this increase is secured by calcium transport systems. In this review, we summarized the involvement of some calcium transport systems in apoptosis, autophagy, necroptosis, ferroptosis, and mitophagy during cancer development and treatment.

  • Czech name

  • Czech description

Classification

  • Type

    J<sub>imp</sub> - Article in a specialist periodical, which is included in the Web of Science database

  • CEP classification

  • OECD FORD branch

    30105 - Physiology (including cytology)

Result continuities

  • Project

    <a href="/en/project/NU21-09-00541" target="_blank" >NU21-09-00541: The role of oxidative stress in the healing of pressure ulcers in patients with spinal cord lesions</a><br>

  • Continuities

    P - Projekt vyzkumu a vyvoje financovany z verejnych zdroju (s odkazem do CEP)

Others

  • Publication year

    2022

  • Confidentiality

    S - Úplné a pravdivé údaje o projektu nepodléhají ochraně podle zvláštních právních předpisů

Data specific for result type

  • Name of the periodical

    Neoplasma

  • ISSN

    0028-2685

  • e-ISSN

    1338-4317

  • Volume of the periodical

    69

  • Issue of the periodical within the volume

    2

  • Country of publishing house

    CZ - CZECH REPUBLIC

  • Number of pages

    10

  • Pages from-to

    264-273

  • UT code for WoS article

    000833596300002

  • EID of the result in the Scopus database

    2-s2.0-85126559559