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Juvenile hormones direct primordial germ cell migration to the embryonic gonad

The result's identifiers

  • Result code in IS VaVaI

    <a href="https://www.isvavai.cz/riv?ss=detail&h=RIV%2F60077344%3A_____%2F24%3A00584656" target="_blank" >RIV/60077344:_____/24:00584656 - isvavai.cz</a>

  • Result on the web

    <a href="https://doi.org/10.1016/j.cub.2023.12.033" target="_blank" >https://doi.org/10.1016/j.cub.2023.12.033</a>

  • DOI - Digital Object Identifier

    <a href="http://dx.doi.org/10.1016/j.cub.2023.12.033" target="_blank" >10.1016/j.cub.2023.12.033</a>

Alternative languages

  • Result language

    angličtina

  • Original language name

    Juvenile hormones direct primordial germ cell migration to the embryonic gonad

  • Original language description

    Germ cells are essential to sexual reproduction. Across the animal kingdom, extracellular signaling isoprenoids, such as retinoic acids (RAs) in vertebrates and juvenile hormones (JHs) in invertebrates, facilitate multiple processes in reproduction. Here we investigated the role of these potent signaling molecules in embryonic germ cell development, using JHs in Drosophila melanogaster as a model system. In contrast to their established endocrine roles during larval and adult germline development, we found that JH signaling acts locally during embryonic development. Using an in vivo biosensor, we observed active JH signaling first within and near primordial germ cells (PGCs) as they migrate to the developing gonad. Through in vivo and in vitro assays, we determined that JHs are both necessary and sufficient for PGC migration. Analysis into the mechanisms of this newly uncovered paracrine JH function revealed that PGC migration was compromised when JHs were decreased or increased, suggesting that specific titers or spatiotemporal JH dynamics are required for robust PGC colonization of the gonad. Compromised PGC migration can impair fertility and cause germ cell tumors in many species, including humans. In mammals, retinoids have many roles in development and reproduction. We found that like JHs in Drosophila, RA was sufficient to impact mouse PGC migration in vitro. Together, our study reveals a previously unanticipated role of isoprenoids as local effectors of pregonadal PGC development and suggests a broadly shared mechanism in PGC migration.

  • Czech name

  • Czech description

Classification

  • Type

    J<sub>imp</sub> - Article in a specialist periodical, which is included in the Web of Science database

  • CEP classification

  • OECD FORD branch

    10608 - Biochemistry and molecular biology

Result continuities

  • Project

    <a href="/en/project/GA22-21244S" target="_blank" >GA22-21244S: Deciphering juvenile hormone (JH) dependent regulation of development and reproduction in mosquitoes using JH null mutants</a><br>

  • Continuities

    I - Institucionalni podpora na dlouhodoby koncepcni rozvoj vyzkumne organizace

Others

  • Publication year

    2024

  • Confidentiality

    S - Úplné a pravdivé údaje o projektu nepodléhají ochraně podle zvláštních právních předpisů

Data specific for result type

  • Name of the periodical

    Current Biology

  • ISSN

    0960-9822

  • e-ISSN

    1879-0445

  • Volume of the periodical

    34

  • Issue of the periodical within the volume

    FEB 5 2024

  • Country of publishing house

    US - UNITED STATES

  • Number of pages

    21

  • Pages from-to

    č. 3

  • UT code for WoS article

    001181652400001

  • EID of the result in the Scopus database

    2-s2.0-85183020212