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The Central Effects of Botulinum Toxin in Dystonia and Spasticity

The result's identifiers

  • Result code in IS VaVaI

    <a href="https://www.isvavai.cz/riv?ss=detail&h=RIV%2F61989592%3A15110%2F21%3A73607315" target="_blank" >RIV/61989592:15110/21:73607315 - isvavai.cz</a>

  • Alternative codes found

    RIV/00098892:_____/21:N0000032

  • Result on the web

    <a href="https://www.mdpi.com/2072-6651/13/2/155" target="_blank" >https://www.mdpi.com/2072-6651/13/2/155</a>

  • DOI - Digital Object Identifier

    <a href="http://dx.doi.org/10.3390/toxins13020155" target="_blank" >10.3390/toxins13020155</a>

Alternative languages

  • Result language

    angličtina

  • Original language name

    The Central Effects of Botulinum Toxin in Dystonia and Spasticity

  • Original language description

    In dystonic and spastic movement disorders, however different in their pathophysiological mechanisms, a similar impairment of sensorimotor control with special emphasis on afferentation is assumed. Peripheral intervention on afferent inputs evokes plastic changes within the central sensorimotor system. Intramuscular application of botulinum toxin type A (BoNT-A) is a standard evidence-based treatment for both conditions. Apart from its peripheral action on muscle spindles, a growing body of evidence suggests that BoNT-A effects could also be mediated by changes at the central level including cerebral cortex. We review recent studies employing electrophysiology and neuroimaging to investigate how intramuscular application of BoNT-A influences cortical reorganization. Based on such data, BoNT-A becomes gradually accepted as a promising tool to correct the maladaptive plastic changes within the sensorimotor cortex. In summary, electrophysiology and especially neuroimaging studies with BoNT-A further our understanding of pathophysiology underlying dystonic and spastic movement disorders and may consequently help develop novel treatment strategies based on neural plasticity

  • Czech name

  • Czech description

Classification

  • Type

    J<sub>imp</sub> - Article in a specialist periodical, which is included in the Web of Science database

  • CEP classification

  • OECD FORD branch

    30210 - Clinical neurology

Result continuities

  • Project

    Result was created during the realization of more than one project. More information in the Projects tab.

  • Continuities

    P - Projekt vyzkumu a vyvoje financovany z verejnych zdroju (s odkazem do CEP)

Others

  • Publication year

    2021

  • Confidentiality

    S - Úplné a pravdivé údaje o projektu nepodléhají ochraně podle zvláštních právních předpisů

Data specific for result type

  • Name of the periodical

    Toxins

  • ISSN

    2072-6651

  • e-ISSN

  • Volume of the periodical

    13

  • Issue of the periodical within the volume

    2

  • Country of publishing house

    CH - SWITZERLAND

  • Number of pages

    21

  • Pages from-to

    "'155(1)'"-"'155(21)'"

  • UT code for WoS article

    000623305700001

  • EID of the result in the Scopus database

    2-s2.0-85102607879