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Polymorphism Rs2421943 of the Insulin-Degrading Enzyme Gene and the Risk of Late-Onset Alzheimer's Disease

The result's identifiers

  • Result code in IS VaVaI

    <a href="https://www.isvavai.cz/riv?ss=detail&h=RIV%2F67985904%3A_____%2F22%3A00560126" target="_blank" >RIV/67985904:_____/22:00560126 - isvavai.cz</a>

  • Alternative codes found

    RIV/00216224:14310/22:00127713

  • Result on the web

    <a href="https://asep.lib.cas.cz/arl-cav/cs/csg/?repo=crepo1&key=38706842811" target="_blank" >https://asep.lib.cas.cz/arl-cav/cs/csg/?repo=crepo1&key=38706842811</a>

  • DOI - Digital Object Identifier

    <a href="http://dx.doi.org/10.2174/1567205019666220302120950" target="_blank" >10.2174/1567205019666220302120950</a>

Alternative languages

  • Result language

    angličtina

  • Original language name

    Polymorphism Rs2421943 of the Insulin-Degrading Enzyme Gene and the Risk of Late-Onset Alzheimer's Disease

  • Original language description

    Background: Insulin-degrading enzyme (IDE) is a widely distributed Zn2+-binding metalloprotease that cleaves multiple short and medium-sized peptides prone to form beta-structures. These include insulin and amyloid-beta peptides. Accumulation and fibrillation of amyloid-beta peptides leading to the formation of amyloid plaques is a characteristic sign of Alzheimer's disease (AD) pathology.nObjective: The study investigated the rs2421943 single nucleotide polymorphism (SNP) of the IDE gene as a risk factor for MCI (mild cognitive impairment) and AD.nMethods: Two independent groups of 1670 patients and controls were included. The AD group consisted of 595 patients and 400 controls, the MCI group involved 135 patients and 540 matched controls. PCR and restriction fragment length analysis were used to analyze the rs2421943 polymorphism. Using the miRBase and RNA22 prediction tools in silico indicated that the rs2421943 polymorphism is a potential target for a specific miRNA (hsa-miR-7110-5p).nResults: AG and GG genotypes of rs2421943 significantly increased the risk of AD, and the AG genotype increased the risk of MCI. It seems the G allele both increases the risk of AD and accelerates the transition through the MCI phase. In silico study revealed that rs2421943 is inside the sequence binding miRNA hsa-miR-7110-5p. The polymorphism could affect the rate of IDE pre-RNA (heterogeneous nuclear RNA, hnRNA) processing, resulting in slower translation, lower levels of IDE, deficient removal of amyloid-beta fragments, and greater risk of and/or accelerated progression of AD.nConclusion: GG and AG genotypes of the single nucleotide polymorphism rs2421943 of insulin-degrading enzyme gene increase the risk of AD and MCI.

  • Czech name

  • Czech description

Classification

  • Type

    J<sub>imp</sub> - Article in a specialist periodical, which is included in the Web of Science database

  • CEP classification

  • OECD FORD branch

    30103 - Neurosciences (including psychophysiology)

Result continuities

  • Project

    <a href="/en/project/NV18-04-00455" target="_blank" >NV18-04-00455: The role of CD36 gene in pathogenesis of Alzheimer´s disease</a><br>

  • Continuities

    I - Institucionalni podpora na dlouhodoby koncepcni rozvoj vyzkumne organizace

Others

  • Publication year

    2022

  • Confidentiality

    S - Úplné a pravdivé údaje o projektu nepodléhají ochraně podle zvláštních právních předpisů

Data specific for result type

  • Name of the periodical

    Current Alzheimer Research

  • ISSN

    1567-2050

  • e-ISSN

    1875-5828

  • Volume of the periodical

    19

  • Issue of the periodical within the volume

    3

  • Country of publishing house

    AE - UNITED ARAB EMIRATES

  • Number of pages

    10

  • Pages from-to

    236-245

  • UT code for WoS article

    000836179200006

  • EID of the result in the Scopus database

    2-s2.0-85128634714