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Association between polymorphism rs2421943 of the insulin-degrading enzyme and schizophrenia: Preliminary report

The result's identifiers

  • Result code in IS VaVaI

    <a href="https://www.isvavai.cz/riv?ss=detail&h=RIV%2F67985904%3A_____%2F23%3A00576044" target="_blank" >RIV/67985904:_____/23:00576044 - isvavai.cz</a>

  • Alternative codes found

    RIV/00216224:14310/23:00134349 RIV/61989592:15110/23:73621295

  • Result on the web

    <a href="https://onlinelibrary.wiley.com/doi/10.1002/jcla.24949" target="_blank" >https://onlinelibrary.wiley.com/doi/10.1002/jcla.24949</a>

  • DOI - Digital Object Identifier

    <a href="http://dx.doi.org/10.1002/jcla.24949" target="_blank" >10.1002/jcla.24949</a>

Alternative languages

  • Result language

    angličtina

  • Original language name

    Association between polymorphism rs2421943 of the insulin-degrading enzyme and schizophrenia: Preliminary report

  • Original language description

    BackgroundInsulin-degrading enzyme (IDE) is an important gene in studies of the pathophysiology of type 2 diabetes mellitus (T2DM). Recent studies have suggested a possible link between type 2 diabetes mellitus (T2DM) and the pathophysiology of schizophrenia (SZ). At the same time, significant changes in insulin-degrading enzyme (IDE) gene expression have been found in the brains of people with schizophrenia. These findings highlight the need to further investigate the role of IDE in schizophrenia pathogenesis. MethodsWe enrolled 733 participants from the Czech Republic, including 383 patients with schizophrenia and 350 healthy controls. Our study focused on the single nucleotide polymorphism (SNP) rs2421943 in the IDE gene, which has previously been associated with the pathogenesis of Alzheimer's disease. The SNP was analyzed using the PCR-RFLP method. ResultsThe G allele of the rs2421943 polymorphism was found to significantly increase the risk of developing SZ (p < 0.01) when a gender-based analysis showed that both AG and GG genotypes were associated with a more than 1.55 times increased risk of SZ in females (p < 0.03) but not in males. Besides, we identified a potential binding site at the G allele locus for has-miR-7110-5p, providing a potential mechanism for the observed association. ConclusionOur results confirm the role of the IDE gene in schizophrenia pathogenesis and suggest that future research should investigate the relationship between miRNA and estrogen influence on IDE expression in schizophrenia pathogenesis.

  • Czech name

  • Czech description

Classification

  • Type

    J<sub>imp</sub> - Article in a specialist periodical, which is included in the Web of Science database

  • CEP classification

  • OECD FORD branch

    30210 - Clinical neurology

Result continuities

  • Project

    Result was created during the realization of more than one project. More information in the Projects tab.

  • Continuities

    P - Projekt vyzkumu a vyvoje financovany z verejnych zdroju (s odkazem do CEP)

Others

  • Publication year

    2023

  • Confidentiality

    S - Úplné a pravdivé údaje o projektu nepodléhají ochraně podle zvláštních právních předpisů

Data specific for result type

  • Name of the periodical

    Journal of Clinical Laboratory Analysis

  • ISSN

    0887-8013

  • e-ISSN

    1098-2825

  • Volume of the periodical

    37

  • Issue of the periodical within the volume

    13-14

  • Country of publishing house

    US - UNITED STATES

  • Number of pages

    7

  • Pages from-to

    e24949

  • UT code for WoS article

    001036281600001

  • EID of the result in the Scopus database

    2-s2.0-85166405685