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A retrotransposon-driven Dicer isoform directs endogenous small interfering RNA production in mouse oocytes

The result's identifiers

  • Result code in IS VaVaI

    <a href="https://www.isvavai.cz/riv?ss=detail&h=RIV%2F68378050%3A_____%2F13%3A00422928" target="_blank" >RIV/68378050:_____/13:00422928 - isvavai.cz</a>

  • Result on the web

    <a href="http://dx.doi.org/10.1016/j.cell.2013.10.001" target="_blank" >http://dx.doi.org/10.1016/j.cell.2013.10.001</a>

  • DOI - Digital Object Identifier

    <a href="http://dx.doi.org/10.1016/j.cell.2013.10.001" target="_blank" >10.1016/j.cell.2013.10.001</a>

Alternative languages

  • Result language

    angličtina

  • Original language name

    A retrotransposon-driven Dicer isoform directs endogenous small interfering RNA production in mouse oocytes

  • Original language description

    In mammals, a single Dicer participates in biogenesis of small RNAs in microRNA (miRNA) and RNAi pathways. In mice, endogenous RNAi is highly active in oocytes, but not in somatic cells, which we ascribe here to an oocyte-specific Dicer isoform (Dicer(O)). Dicer(O) lacks the N-terminal DExD helicase domain and has higher cleavage activity than the full-length Dicer in somatic cells (Dicer(S)). Unlike Dicer(S), Dicer(O) efficiently produces small RNAs from long double-stranded (dsRNA) substrates. Expression of the Dicer(O) isoform is driven by an intronic MT-C retrotransposon promoter, deletion of which causes loss of Dicer(O) and female sterility. Oocytes from females lacking the MT-C element show meiotic spindle defects and increased levels of endogenous small interfering RNA (endo-siRNA) targets, phenocopying the maternal Dicer null phenotype. The alternative Dicer isoform, whose phylogenetic origin demonstrates evolutionary plasticity of RNA-silencing pathways, is the main determina

  • Czech name

  • Czech description

Classification

  • Type

    J<sub>x</sub> - Unclassified - Peer-reviewed scientific article (Jimp, Jsc and Jost)

  • CEP classification

    EB - Genetics and molecular biology

  • OECD FORD branch

Result continuities

  • Project

    Result was created during the realization of more than one project. More information in the Projects tab.

  • Continuities

    I - Institucionalni podpora na dlouhodoby koncepcni rozvoj vyzkumne organizace

Others

  • Publication year

    2013

  • Confidentiality

    S - Úplné a pravdivé údaje o projektu nepodléhají ochraně podle zvláštních právních předpisů

Data specific for result type

  • Name of the periodical

    Cell

  • ISSN

    0092-8674

  • e-ISSN

  • Volume of the periodical

    155

  • Issue of the periodical within the volume

    4

  • Country of publishing house

    US - UNITED STATES

  • Number of pages

    10

  • Pages from-to

    807-816

  • UT code for WoS article

    000326934300011

  • EID of the result in the Scopus database