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Cep97 Is Required for Centriole Structural Integrity and Cilia Formation in Drosophila

The result's identifiers

  • Result code in IS VaVaI

    <a href="https://www.isvavai.cz/riv?ss=detail&h=RIV%2F68378050%3A_____%2F20%3A00531953" target="_blank" >RIV/68378050:_____/20:00531953 - isvavai.cz</a>

  • Result on the web

    <a href="https://www.cell.com/current-biology/fulltext/S0960-9822(20)30765-X?_returnURL=https%3A%2F%2Flinkinghub.elsevier.com%2Fretrieve%2Fpii%2FS096098222030765X%3Fshowall%3Dtrue" target="_blank" >https://www.cell.com/current-biology/fulltext/S0960-9822(20)30765-X?_returnURL=https%3A%2F%2Flinkinghub.elsevier.com%2Fretrieve%2Fpii%2FS096098222030765X%3Fshowall%3Dtrue</a>

  • DOI - Digital Object Identifier

    <a href="http://dx.doi.org/10.1016/j.cub.2020.05.078" target="_blank" >10.1016/j.cub.2020.05.078</a>

Alternative languages

  • Result language

    angličtina

  • Original language name

    Cep97 Is Required for Centriole Structural Integrity and Cilia Formation in Drosophila

  • Original language description

    Centrioles are highly elaborate microtubule-based structures responsible for the formation of centrosomes and cilia. Despite considerable variation across species and tissues within any given tissue, their size is essentially constant [1, 2]. While the diameter of the centriole cylinder is set by the dimensions of the inner scaffolding structure of the cartwheel [3], how centriole length is set so precisely and stably maintained over many cell divisions is not well understood. Cep97 and CP110 are conserved proteins that localize to the distal end of centrioles and have been reported to limit centriole elongation in vertebrates [4, 5]. Here, we examine Cep97 function in Drosophila melanogaster. We show that Cep97 is essential for formation of full-length centrioles in multiple tissues of the fly. We further identify the microtubule deacetylase Sirt2 as a Cep97 interactor. Deletion of Sirt2 likewise affects centriole size. Interestingly, so does deletion of the acetylase Atat1, indicating that loss of stabilizing acetylmarks impairs centriole integrity. Cep97 and CP110 were originally identified as inhibitors of cilia formation in vertebrate cultured cells [6], and loss of CP110 is a widely used marker of basal body maturation. In contrast, in Drosophila, Cep97 appears to be only transiently removed from basal bodies and loss of Cep97 strongly impairs ciliogenesis. Collectively, our results support a model whereby Cep97 functions as part of a protective cap that acts together with the microtubule acetylation machinery to maintain centriole stability, essential for proper function in cilium biogenesis.

  • Czech name

  • Czech description

Classification

  • Type

    J<sub>imp</sub> - Article in a specialist periodical, which is included in the Web of Science database

  • CEP classification

  • OECD FORD branch

    10601 - Cell biology

Result continuities

  • Project

    <a href="/en/project/GJ17-20613Y" target="_blank" >GJ17-20613Y: BBSome and actin interplay in ciliogenesis and formation of the immunological synapse</a><br>

  • Continuities

    I - Institucionalni podpora na dlouhodoby koncepcni rozvoj vyzkumne organizace

Others

  • Publication year

    2020

  • Confidentiality

    S - Úplné a pravdivé údaje o projektu nepodléhají ochraně podle zvláštních právních předpisů

Data specific for result type

  • Name of the periodical

    Current Biology

  • ISSN

    0960-9822

  • e-ISSN

  • Volume of the periodical

    30

  • Issue of the periodical within the volume

    15

  • Country of publishing house

    US - UNITED STATES

  • Number of pages

    12

  • Pages from-to

    3045-3056

  • UT code for WoS article

    000555598100019

  • EID of the result in the Scopus database