Helicobacter pylori ? Not Only a Gastric Pathogene?
Identifikátory výsledku
Kód výsledku v IS VaVaI
<a href="https://www.isvavai.cz/riv?ss=detail&h=RIV%2F00216208%3A11110%2F11%3A9192" target="_blank" >RIV/00216208:11110/11:9192 - isvavai.cz</a>
Nalezeny alternativní kódy
RIV/00064203:_____/11:7478 RIV/00064165:_____/11:9192
Výsledek na webu
<a href="http://www.intechweb.org/books/show/title/peptic-ulcer-disease" target="_blank" >http://www.intechweb.org/books/show/title/peptic-ulcer-disease</a>
DOI - Digital Object Identifier
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Alternativní jazyky
Jazyk výsledku
angličtina
Název v původním jazyce
Helicobacter pylori ? Not Only a Gastric Pathogene?
Popis výsledku v původním jazyce
Recent studies indicate that Helicobacter pylori (HP) may exist in the oropharynx independently to the gastric infection. Comparison of genotypes of HP in the oral cavity, oropharynx, and stomach prooved that an individual can host more than one strain in various locations. Differences were found in the presence of cagA gene and in the structure of vacA gene. The findings of HP in the oral cavity and oropharynx without demonstrable specific anti-HP antibodies in serum are remarkable. This could be explained by an early detection of HP after primary infection, when the antibody response has not started yet. HP could also colonize the tisssues without inducing the host immune response. Another explanation is the presence of HP coccoid forms. The questionof transmission of HP has not been satisfactorily resolved yet. Changes in the expression of some cytokines caused by HP were described in the gastric mucosa and were associated with malignit transformation. Similar mechanisms can be exp
Název v anglickém jazyce
Helicobacter pylori ? Not Only a Gastric Pathogene?
Popis výsledku anglicky
Recent studies indicate that Helicobacter pylori (HP) may exist in the oropharynx independently to the gastric infection. Comparison of genotypes of HP in the oral cavity, oropharynx, and stomach prooved that an individual can host more than one strain in various locations. Differences were found in the presence of cagA gene and in the structure of vacA gene. The findings of HP in the oral cavity and oropharynx without demonstrable specific anti-HP antibodies in serum are remarkable. This could be explained by an early detection of HP after primary infection, when the antibody response has not started yet. HP could also colonize the tisssues without inducing the host immune response. Another explanation is the presence of HP coccoid forms. The questionof transmission of HP has not been satisfactorily resolved yet. Changes in the expression of some cytokines caused by HP were described in the gastric mucosa and were associated with malignit transformation. Similar mechanisms can be exp
Klasifikace
Druh
C - Kapitola v odborné knize
CEP obor
FF - ORL, oftalmologie, stomatologie
OECD FORD obor
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Návaznosti výsledku
Projekt
<a href="/cs/project/NT11523" target="_blank" >NT11523: Vztah infekce Helicobacter pylori a gastroesophageálního reflexu k etiopatogenezi orofaryngeálních a laryngálních nádorů</a><br>
Návaznosti
I - Institucionalni podpora na dlouhodoby koncepcni rozvoj vyzkumne organizace
Ostatní
Rok uplatnění
2011
Kód důvěrnosti údajů
S - Úplné a pravdivé údaje o projektu nepodléhají ochraně podle zvláštních právních předpisů
Údaje specifické pro druh výsledku
Název knihy nebo sborníku
Peptic Ulcer Disease
ISBN
978-953-307-976-9
Počet stran výsledku
12
Strana od-do
99-110
Počet stran knihy
482
Název nakladatele
InTech
Místo vydání
Rijeka
Kód UT WoS kapitoly
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