Hemodynamic changes in children with hypercapnic respiratory failure during respiratory syncytial virus infection

Kód výsledku v IS VaVaI

<a href="https://www.isvavai.cz/riv?ss=detail&h=RIV%2F00216208%3A11140%2F15%3A10227306" target="_blank" >RIV/00216208:11140/15:10227306 - isvavai.cz</a>

Výsledek na webu

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DOI - Digital Object Identifier

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Jazyk výsledku

angličtina

Název v původním jazyce

Hemodynamic changes in children with hypercapnic respiratory failure during respiratory syncytial virus infection

Popis výsledku v původním jazyce

Acute bronchiolitis associated with Respiratory Syncytial Virus (RSV) infection is an example of progressive hypercapnic hypoventilation. Acute bronchiolitis is the most difficult course of the disease, and lower airways' obstruction is difficult to control. Typical symptoms are progressive tachypnoea, grunting, chest retraction, increased difficulty in breathing, hypoventilation, and mixed dyspnoea, all of which are resistant to bronchodilatory or anti-inflammatory therapy. Increased airway resistancecauses parallel hyperinflation and alveoli consolidation. These pulmonary pathophysiological changes are a source of such complications as pneumonia, fluidothorax, and others. Severe pulmonary pathology causes cardiopupmonary interaction that has an adverse impact on blood circulation. The consequences of such interaction are right ventricle pressure overload, a reduction in pulmonary flow, and a decrease in cardiac output. This situation has caused complications and limits the possibili

Název v anglickém jazyce

Hemodynamic changes in children with hypercapnic respiratory failure during respiratory syncytial virus infection

Popis výsledku anglicky

Acute bronchiolitis associated with Respiratory Syncytial Virus (RSV) infection is an example of progressive hypercapnic hypoventilation. Acute bronchiolitis is the most difficult course of the disease, and lower airways' obstruction is difficult to control. Typical symptoms are progressive tachypnoea, grunting, chest retraction, increased difficulty in breathing, hypoventilation, and mixed dyspnoea, all of which are resistant to bronchodilatory or anti-inflammatory therapy. Increased airway resistancecauses parallel hyperinflation and alveoli consolidation. These pulmonary pathophysiological changes are a source of such complications as pneumonia, fluidothorax, and others. Severe pulmonary pathology causes cardiopupmonary interaction that has an adverse impact on blood circulation. The consequences of such interaction are right ventricle pressure overload, a reduction in pulmonary flow, and a decrease in cardiac output. This situation has caused complications and limits the possibili

Druh

J_x - Nezařazeno - Článek v odborném periodiku (Jimp, Jsc a Jost)

CEP obor

FG - Pediatrie

OECD FORD obor

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Projekt

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Návaznosti

Z - Vyzkumny zamer (s odkazem do CEZ)<br>S - Specificky vyzkum na vysokych skolach<br>I - Institucionalni podpora na dlouhodoby koncepcni rozvoj vyzkumne organizace

Rok uplatnění

2015

Kód důvěrnosti údajů

S - Úplné a pravdivé údaje o projektu nepodléhají ochraně podle zvláštních právních předpisů

Název periodika

Plzeňský lékařský sborník

ISSN

0551-1038

e-ISSN

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Svazek periodika

2014

Číslo periodika v rámci svazku

80

Stát vydavatele periodika

CZ - Česká republika

Počet stran výsledku

11

Strana od-do

9-19

Kód UT WoS článku

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EID výsledku v databázi Scopus

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