A review on fatty liver disease or non-alcoholic fatty liver disease and hepatocellular carcinoma
Identifikátory výsledku
Kód výsledku v IS VaVaI
<a href="https://www.isvavai.cz/riv?ss=detail&h=RIV%2F00843989%3A_____%2F21%3AE0109546" target="_blank" >RIV/00843989:_____/21:E0109546 - isvavai.cz</a>
Nalezeny alternativní kódy
RIV/61988987:17110/21:A2302CIC
Výsledek na webu
<a href="https://worldresearchersassociations.com/Archives/RJBT/Vol(16)2021//October%202021/A%20review%20on%20fatty%20liver%20disease%20or%20non-alcoholic.pdf" target="_blank" >https://worldresearchersassociations.com/Archives/RJBT/Vol(16)2021//October%202021/A%20review%20on%20fatty%20liver%20disease%20or%20non-alcoholic.pdf</a>
DOI - Digital Object Identifier
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Alternativní jazyky
Jazyk výsledku
angličtina
Název v původním jazyce
A review on fatty liver disease or non-alcoholic fatty liver disease and hepatocellular carcinoma
Popis výsledku v původním jazyce
The non-alcoholic fatty liver disease (NAFLD) is the predominant etiological factor for liver disease. There is a risk of the development of hepatocellular carcinoma (HCC) in patients suffering from NAFLD. Non-alcoholic steatohepatitis (NASH) is one of the risk factors for the development of HCC. The aim is to discuss an association of NAFLD and HCC in the adult population. HCC is one of the debilitating complications of NAFLD/NASH and obesity is a causative factor for NAFLD/NASH. Various clinical data suggest that obesity appears to be a causative factor in the progression of NAFLD/NASH to HCC. We searched data from the PubMed/Medline and Google Scholar databases including various studies and review articles. Significantly, an increased number of HCC patients with cryptogenic liver disease had well-differentiated tumors than in HCC patients with chronic viral hepatitis and alcoholism. HCC is one of the debilitating complications of NAFLD/NASH and obesity is a causative factor for NAFLD/NASH. Various preclinical and clinical data suggest that obesity appears to be an important causative factor in the progression of NAFLD/NASH to HCC.
Název v anglickém jazyce
A review on fatty liver disease or non-alcoholic fatty liver disease and hepatocellular carcinoma
Popis výsledku anglicky
The non-alcoholic fatty liver disease (NAFLD) is the predominant etiological factor for liver disease. There is a risk of the development of hepatocellular carcinoma (HCC) in patients suffering from NAFLD. Non-alcoholic steatohepatitis (NASH) is one of the risk factors for the development of HCC. The aim is to discuss an association of NAFLD and HCC in the adult population. HCC is one of the debilitating complications of NAFLD/NASH and obesity is a causative factor for NAFLD/NASH. Various clinical data suggest that obesity appears to be a causative factor in the progression of NAFLD/NASH to HCC. We searched data from the PubMed/Medline and Google Scholar databases including various studies and review articles. Significantly, an increased number of HCC patients with cryptogenic liver disease had well-differentiated tumors than in HCC patients with chronic viral hepatitis and alcoholism. HCC is one of the debilitating complications of NAFLD/NASH and obesity is a causative factor for NAFLD/NASH. Various preclinical and clinical data suggest that obesity appears to be an important causative factor in the progression of NAFLD/NASH to HCC.
Klasifikace
Druh
J<sub>imp</sub> - Článek v periodiku v databázi Web of Science
CEP obor
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OECD FORD obor
30212 - Surgery
Návaznosti výsledku
Projekt
—
Návaznosti
V - Vyzkumna aktivita podporovana z jinych verejnych zdroju
Ostatní
Rok uplatnění
2021
Kód důvěrnosti údajů
S - Úplné a pravdivé údaje o projektu nepodléhají ochraně podle zvláštních právních předpisů
Údaje specifické pro druh výsledku
Název periodika
Research journal of biotechnology
ISSN
2278-4535
e-ISSN
2278-4535
Svazek periodika
16
Číslo periodika v rámci svazku
10
Stát vydavatele periodika
IN - Indická republika
Počet stran výsledku
7
Strana od-do
156-162
Kód UT WoS článku
000702072800020
EID výsledku v databázi Scopus
2-s2.0-85115745963