An Appraisal of Methods Recently Recommended for Testing Salt Sensitivity of Blood Pressure
Identifikátory výsledku
Kód výsledku v IS VaVaI
<a href="https://www.isvavai.cz/riv?ss=detail&h=RIV%2F67985823%3A_____%2F17%3A00474740" target="_blank" >RIV/67985823:_____/17:00474740 - isvavai.cz</a>
Výsledek na webu
<a href="http://dx.doi.org/10.1161/JAHA.117.005653" target="_blank" >http://dx.doi.org/10.1161/JAHA.117.005653</a>
DOI - Digital Object Identifier
<a href="http://dx.doi.org/10.1161/JAHA.117.005653" target="_blank" >10.1161/JAHA.117.005653</a>
Alternativní jazyky
Jazyk výsledku
angličtina
Název v původním jazyce
An Appraisal of Methods Recently Recommended for Testing Salt Sensitivity of Blood Pressure
Popis výsledku v původním jazyce
It is widely held that in response to high salt diets, normal individuals are acutely and chronically resistant to salt-induced hypertension because they rapidly excrete salt and retain little of it, such that their blood volume, and therefore blood pressure, does not increase. Conversely, it is also widely held that salt-sensitive individuals develop salt-induced hypertension because of an impaired renal capacity to excrete salt that causes greater salt retention and blood volume expansion than that which occurs in normal salt-resistant individuals. Here we review results of both acute and chronic salt-loading studies that have compared salt-induced changes in sodium retention and blood volume between normal subjects (salt-resistant normotensive controls) and salt-sensitive subjects.
Název v anglickém jazyce
An Appraisal of Methods Recently Recommended for Testing Salt Sensitivity of Blood Pressure
Popis výsledku anglicky
It is widely held that in response to high salt diets, normal individuals are acutely and chronically resistant to salt-induced hypertension because they rapidly excrete salt and retain little of it, such that their blood volume, and therefore blood pressure, does not increase. Conversely, it is also widely held that salt-sensitive individuals develop salt-induced hypertension because of an impaired renal capacity to excrete salt that causes greater salt retention and blood volume expansion than that which occurs in normal salt-resistant individuals. Here we review results of both acute and chronic salt-loading studies that have compared salt-induced changes in sodium retention and blood volume between normal subjects (salt-resistant normotensive controls) and salt-sensitive subjects.
Klasifikace
Druh
J<sub>imp</sub> - Článek v periodiku v databázi Web of Science
CEP obor
—
OECD FORD obor
30201 - Cardiac and Cardiovascular systems
Návaznosti výsledku
Projekt
—
Návaznosti
I - Institucionalni podpora na dlouhodoby koncepcni rozvoj vyzkumne organizace
Ostatní
Rok uplatnění
2017
Kód důvěrnosti údajů
S - Úplné a pravdivé údaje o projektu nepodléhají ochraně podle zvláštních právních předpisů
Údaje specifické pro druh výsledku
Název periodika
Journal of the American Heart Association
ISSN
2047-9980
e-ISSN
—
Svazek periodika
6
Číslo periodika v rámci svazku
4
Stát vydavatele periodika
US - Spojené státy americké
Počet stran výsledku
8
Strana od-do
—
Kód UT WoS článku
000404098500059
EID výsledku v databázi Scopus
2-s2.0-85016635385