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Bleomycin-induced chromosomal damage and shortening of telomeres in peripheral blood lymphocytes of incident cancer patients

The result's identifiers

  • Result code in IS VaVaI

    <a href="https://www.isvavai.cz/riv?ss=detail&h=RIV%2F00216208%3A11110%2F18%3A10363757" target="_blank" >RIV/00216208:11110/18:10363757 - isvavai.cz</a>

  • Alternative codes found

    RIV/68378041:_____/18:00480900 RIV/00216208:11120/18:43915697 RIV/00216208:11140/18:10363757 RIV/00064190:_____/18:N0000036 RIV/00064165:_____/18:10363757

  • Result on the web

    <a href="http://dx.doi.org/10.1002/gcc.22508" target="_blank" >http://dx.doi.org/10.1002/gcc.22508</a>

  • DOI - Digital Object Identifier

    <a href="http://dx.doi.org/10.1002/gcc.22508" target="_blank" >10.1002/gcc.22508</a>

Alternative languages

  • Result language

    angličtina

  • Original language name

    Bleomycin-induced chromosomal damage and shortening of telomeres in peripheral blood lymphocytes of incident cancer patients

  • Original language description

    Disruption of genomic integrity due to deficient DNA repair capacity and telomere shortening constitute hallmarks of malignant diseases. Incomplete or deficient repair of DNA double-strand breaks (DSB) is manifested by chromosomal aberrations (CAs) and their frequency reflects inter-individual differences of response to exposure to mutagenic compounds. In this study, we investigated chromosomal integrity in peripheral blood lymphocytes (PBL) from newly diagnosed cancer patients, including 47 breast (BC) and 44 colorectal cancer (CRC) patients and 90 matched healthy controls. Mutagen sensitivity was evaluated by measuring chromatid breaks (CTAs) induced by bleomycin and supplemented by the chemiluminescent measurement of γ-H2AX phosphorylation in 19 cancer patients (11 BC, 8 CRC). Relative telomere length (RTL) was determined in 22 BC, 32 CRC and 64 controls. We observed statistically significant increased level of CTAs (p=0.03) and increased percentage of aberrant cells (ACs) with CTAs (p=0.05) in CRC patients compared to controls after bleomycin treatment. No differences were observed between BC cases and corresponding controls. CRC and BC patients with shorter RTL (below median) exhibited significantly higher amount of ACs (p=0.02), CTAs (p=0.02) and cells with high frequency of CTAs (&gt;=12 CTAs/PBL; p=0.03) after bleomycin treatment. No such associations were observed in healthy controls. γ-H2AX phosphorylation after bleomycin treatment in PBL did not differ between CRC and BC patients. Our results suggest that altered DSB repair measured by sensitivity towards mutagen in PBL occurs particularly in CRC carcinogenesis. Irrespective of cancer type, telomere shortening may be associated with a decreased capacity to repair DSB.

  • Czech name

  • Czech description

Classification

  • Type

    J<sub>imp</sub> - Article in a specialist periodical, which is included in the Web of Science database

  • CEP classification

  • OECD FORD branch

    10601 - Cell biology

Result continuities

  • Project

    Result was created during the realization of more than one project. More information in the Projects tab.

  • Continuities

    S - Specificky vyzkum na vysokych skolach<br>I - Institucionalni podpora na dlouhodoby koncepcni rozvoj vyzkumne organizace

Others

  • Publication year

    2018

  • Confidentiality

    S - Úplné a pravdivé údaje o projektu nepodléhají ochraně podle zvláštních právních předpisů

Data specific for result type

  • Name of the periodical

    Genes, Chromosomes &amp; Cancer

  • ISSN

    1045-2257

  • e-ISSN

  • Volume of the periodical

    57

  • Issue of the periodical within the volume

    2

  • Country of publishing house

    US - UNITED STATES

  • Number of pages

    9

  • Pages from-to

    61-69

  • UT code for WoS article

    000418151700002

  • EID of the result in the Scopus database

    2-s2.0-85041101683