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Sprimg viremia of cyprinid species and their hybrids

Identifikátory výsledku

  • Kód výsledku v IS VaVaI

    <a href="https://www.isvavai.cz/riv?ss=detail&h=RIV%2F00216224%3A14310%2F18%3A00101694" target="_blank" >RIV/00216224:14310/18:00101694 - isvavai.cz</a>

  • Výsledek na webu

  • DOI - Digital Object Identifier

Alternativní jazyky

  • Jazyk výsledku

    angličtina

  • Název v původním jazyce

    Sprimg viremia of cyprinid species and their hybrids

  • Popis výsledku v původním jazyce

    Viral diseases in aquacultures are serious problem mainly due to lack of treatment options. Spring viremia of carp (SVC) is caused by Rhabdovirus carpio, a bullet-shaped RNA virus. The disease has been reported in many regions of the world, mainly in Europe, Middle East and also in America. Mortality caused by SVCV has reached 70% in yearling carp from European populations. In our work, we focused on SVCV mechanism, development and progress. We examined the level of expression of SVCV in C. carpio, C. auratus gibelio and their F1 hybrids. Several hypotheses have been predicted for parasite load in parental species and their F1 hybrids (dominance, resistance, susceptibility or additive scenario). The main aim was to investigate the SVCV infection progress in three above mentioned groups of fish. Our preliminary findings about viral expression within host groups correspond with previous studies in this field. In C. gibelio, viral copies rapidly increased in all samples at 24-48 hpi to the maximum. In C. carpio, slower progress of an infection was observed and viral copies increased to maximum at 3-5 dpi in this group. In the hybrid group, the infection progress was very heterogenic between individuals and could not be characterized uniformly. The regulation of SVCV infection may be potentially useful in process of eradication of introduced species, immune response research using breed lines, or in development of fish antivirotics (vaccines or antiviral treatment).

  • Název v anglickém jazyce

    Sprimg viremia of cyprinid species and their hybrids

  • Popis výsledku anglicky

    Viral diseases in aquacultures are serious problem mainly due to lack of treatment options. Spring viremia of carp (SVC) is caused by Rhabdovirus carpio, a bullet-shaped RNA virus. The disease has been reported in many regions of the world, mainly in Europe, Middle East and also in America. Mortality caused by SVCV has reached 70% in yearling carp from European populations. In our work, we focused on SVCV mechanism, development and progress. We examined the level of expression of SVCV in C. carpio, C. auratus gibelio and their F1 hybrids. Several hypotheses have been predicted for parasite load in parental species and their F1 hybrids (dominance, resistance, susceptibility or additive scenario). The main aim was to investigate the SVCV infection progress in three above mentioned groups of fish. Our preliminary findings about viral expression within host groups correspond with previous studies in this field. In C. gibelio, viral copies rapidly increased in all samples at 24-48 hpi to the maximum. In C. carpio, slower progress of an infection was observed and viral copies increased to maximum at 3-5 dpi in this group. In the hybrid group, the infection progress was very heterogenic between individuals and could not be characterized uniformly. The regulation of SVCV infection may be potentially useful in process of eradication of introduced species, immune response research using breed lines, or in development of fish antivirotics (vaccines or antiviral treatment).

Klasifikace

  • Druh

    O - Ostatní výsledky

  • CEP obor

  • OECD FORD obor

    10600 - Biological sciences

Návaznosti výsledku

  • Projekt

    <a href="/cs/project/GBP505%2F12%2FG112" target="_blank" >GBP505/12/G112: ECIP - Evropské centrum ichtyoparazitologie</a><br>

  • Návaznosti

    P - Projekt vyzkumu a vyvoje financovany z verejnych zdroju (s odkazem do CEP)

Ostatní

  • Rok uplatnění

    2018

  • Kód důvěrnosti údajů

    S - Úplné a pravdivé údaje o projektu nepodléhají ochraně podle zvláštních právních předpisů