Mechanisms of Appetite Regulation
Identifikátory výsledku
Kód výsledku v IS VaVaI
<a href="https://www.isvavai.cz/riv?ss=detail&h=RIV%2F00216208%3A11120%2F10%3A00002731" target="_blank" >RIV/00216208:11120/10:00002731 - isvavai.cz</a>
Nalezeny alternativní kódy
RIV/00216208:11130/10:6355 RIV/00064203:_____/10:6355
Výsledek na webu
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DOI - Digital Object Identifier
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Alternativní jazyky
Jazyk výsledku
angličtina
Název v původním jazyce
Mechanisms of Appetite Regulation
Popis výsledku v původním jazyce
Appetite regulation and adequate eating behavior are crucial for survival. To begin and to stop eating is a complex process. Appetite regulation, perception of hunger and satiety, eating behavior, and food preferences are in great part determined by genetic factors. Therefore, if tasty and energy-rich food is freely available in potentially unlimited quantities, overeating may occur due to insufficient defense mechanisms. Pleasure-seeking and edonic responses to food intake are mediated by humoral substances, for example, endorphins, dopamine, and endocannabinoids. In conclusion, a number of signals contribute to the central regulation of appetite and satiety by acting directly on the hypothalamic arcuate nucleus. Mutations of genes involved in energybalance regulation as the leptin-melanocortin pathway lead to a loss of control over appetite and early-onset severe obesity. Probably more factors that play a role in energy intake regulation exist and so far have not been identified.
Název v anglickém jazyce
Mechanisms of Appetite Regulation
Popis výsledku anglicky
Appetite regulation and adequate eating behavior are crucial for survival. To begin and to stop eating is a complex process. Appetite regulation, perception of hunger and satiety, eating behavior, and food preferences are in great part determined by genetic factors. Therefore, if tasty and energy-rich food is freely available in potentially unlimited quantities, overeating may occur due to insufficient defense mechanisms. Pleasure-seeking and edonic responses to food intake are mediated by humoral substances, for example, endorphins, dopamine, and endocannabinoids. In conclusion, a number of signals contribute to the central regulation of appetite and satiety by acting directly on the hypothalamic arcuate nucleus. Mutations of genes involved in energybalance regulation as the leptin-melanocortin pathway lead to a loss of control over appetite and early-onset severe obesity. Probably more factors that play a role in energy intake regulation exist and so far have not been identified.
Klasifikace
Druh
J<sub>x</sub> - Nezařazeno - Článek v odborném periodiku (Jimp, Jsc a Jost)
CEP obor
FB - Endokrinologie, diabetologie, metabolismus, výživa
OECD FORD obor
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Návaznosti výsledku
Projekt
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Návaznosti
Z - Vyzkumny zamer (s odkazem do CEZ)
Ostatní
Rok uplatnění
2010
Kód důvěrnosti údajů
S - Úplné a pravdivé údaje o projektu nepodléhají ochraně podle zvláštních právních předpisů
Údaje specifické pro druh výsledku
Název periodika
Journal of Pediatric Gastroenterology and Nutrition
ISSN
0277-2116
e-ISSN
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Svazek periodika
51
Číslo periodika v rámci svazku
Suppl. 3
Stát vydavatele periodika
US - Spojené státy americké
Počet stran výsledku
2
Strana od-do
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Kód UT WoS článku
000284810100005
EID výsledku v databázi Scopus
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