Hetero-interface-engineered sulfur vacancy and oxygen doping in hollow Co9S8/Fe7S8 nanospheres towards monopersulfate activation for boosting intrinsic electron transfer in paracetamol degradation

Kód výsledku v IS VaVaI

<a href="https://www.isvavai.cz/riv?ss=detail&h=RIV%2F46747885%3A24620%2F23%3A00011197" target="_blank" >RIV/46747885:24620/23:00011197 - isvavai.cz</a>

Výsledek na webu

<a href="https://www.sciencedirect.com/science/article/pii/S0926337323001935" target="_blank" >https://www.sciencedirect.com/science/article/pii/S0926337323001935</a>

DOI - Digital Object Identifier

<a href="http://dx.doi.org/10.1016/j.apcatb.2023.122550" target="_blank" >10.1016/j.apcatb.2023.122550</a>

Jazyk výsledku

angličtina

Název v původním jazyce

Hetero-interface-engineered sulfur vacancy and oxygen doping in hollow Co9S8/Fe7S8 nanospheres towards monopersulfate activation for boosting intrinsic electron transfer in paracetamol degradation

Popis výsledku v původním jazyce

Designing defects-rich hollow heterostructure bimetal sulfides is considered as an efficient strategy for accelerated monopersulfate (MPS) activation. Herein, mono-step sulfidation was employed to develop sulfur vacancy (SV)-rich hollow oxygen-doped Co9S8/Fe7S8 (O-CSFS). SV and oxygen doping-induced highly electroactive sites, low charge resistance, and increased conductivity of O-CSFS accounted for its superior performance. Reactive oxygen species (ROS)-driven pathway and electron transfer (ET)-driven pathway were revealed to be responsible for PCM degradation in O-CSFS/MPS system, but the role of ET-driven pathway was more significant. The ROS-driven pathway was mainly attributed to electrons-rich low valance of Co atoms which activated MPS to generate different ROS without •OH contribution and with a greater role of SO4•− than 1O2. Doped O, S species, and surface-active O-CSFS/MPS complex in ET-driven pathway, meanwhile, acquired electrons from PCM, resulting in enhanced PCM oxidation. This study provided more insight into ET-enhanced efficient PCM degradation induced by SV and oxygen-doping.

Název v anglickém jazyce

Hetero-interface-engineered sulfur vacancy and oxygen doping in hollow Co9S8/Fe7S8 nanospheres towards monopersulfate activation for boosting intrinsic electron transfer in paracetamol degradation

Popis výsledku anglicky

Designing defects-rich hollow heterostructure bimetal sulfides is considered as an efficient strategy for accelerated monopersulfate (MPS) activation. Herein, mono-step sulfidation was employed to develop sulfur vacancy (SV)-rich hollow oxygen-doped Co9S8/Fe7S8 (O-CSFS). SV and oxygen doping-induced highly electroactive sites, low charge resistance, and increased conductivity of O-CSFS accounted for its superior performance. Reactive oxygen species (ROS)-driven pathway and electron transfer (ET)-driven pathway were revealed to be responsible for PCM degradation in O-CSFS/MPS system, but the role of ET-driven pathway was more significant. The ROS-driven pathway was mainly attributed to electrons-rich low valance of Co atoms which activated MPS to generate different ROS without •OH contribution and with a greater role of SO4•− than 1O2. Doped O, S species, and surface-active O-CSFS/MPS complex in ET-driven pathway, meanwhile, acquired electrons from PCM, resulting in enhanced PCM oxidation. This study provided more insight into ET-enhanced efficient PCM degradation induced by SV and oxygen-doping.

Druh

J_imp - Článek v periodiku v databázi Web of Science

CEP obor

—

OECD FORD obor

20402 - Chemical process engineering

Projekt

—

Návaznosti

I - Institucionalni podpora na dlouhodoby koncepcni rozvoj vyzkumne organizace

Rok uplatnění

2023

Kód důvěrnosti údajů

S - Úplné a pravdivé údaje o projektu nepodléhají ochraně podle zvláštních právních předpisů

Název periodika

APPLIED CATALYSIS B-ENVIRONMENTAL>

ISSN

0926-3373

e-ISSN

—

Svazek periodika

330

Číslo periodika v rámci svazku

AUG 5

Stát vydavatele periodika

NL - Nizozemsko

Počet stran výsledku

23

Strana od-do

—

Kód UT WoS článku

001088424900001

EID výsledku v databázi Scopus

2-s2.0-85150063071