Cancer and Structure

Kód výsledku v IS VaVaI

<a href="https://www.isvavai.cz/riv?ss=detail&h=RIV%2F60461373%3A22340%2F18%3A43916388" target="_blank" >RIV/60461373:22340/18:43916388 - isvavai.cz</a>

Výsledek na webu

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DOI - Digital Object Identifier

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Jazyk výsledku

angličtina

Název v původním jazyce

Cancer and Structure

Popis výsledku v původním jazyce

We consider the principle loss of cell structure (information) caused by normal cell division. This loss or aging by a &apos;tooth of time‘ can be measured by information entropy. Functionally, it can be described by a carnotized model of information trans¬fer, by a direct heat cycle. The growth of thermodynamic entropy in its environment is similar to this loss, measured, in biology, by the shortenning the length of cell telomere. Telomere is, approximatelly, a box of chromozomes and its length codes the age of the cell, or, the number of the cell’s predecessors in the normal situation. During pathological cell proliferation the opposite situation arises. Cells with a precise structure (of a different type) are generated now. This growth of cells‘ structure in a certain locality (within the whole organism) is ‘paid for‘ by draining off energy from its environment. Again, this situation is describable by the thermodynamic model of information transfer, but in a reverse form this time. The decreasing value of thermodynamic entropy or increasing value of information entropy is now evidenced locally with the higher growth of the thermodynamic entropy in the environment. The growth of cell structure is now measured by the lengthening of their telomeres.

Název v anglickém jazyce

Cancer and Structure

Popis výsledku anglicky

We consider the principle loss of cell structure (information) caused by normal cell division. This loss or aging by a &apos;tooth of time‘ can be measured by information entropy. Functionally, it can be described by a carnotized model of information trans¬fer, by a direct heat cycle. The growth of thermodynamic entropy in its environment is similar to this loss, measured, in biology, by the shortenning the length of cell telomere. Telomere is, approximatelly, a box of chromozomes and its length codes the age of the cell, or, the number of the cell’s predecessors in the normal situation. During pathological cell proliferation the opposite situation arises. Cells with a precise structure (of a different type) are generated now. This growth of cells‘ structure in a certain locality (within the whole organism) is ‘paid for‘ by draining off energy from its environment. Again, this situation is describable by the thermodynamic model of information transfer, but in a reverse form this time. The decreasing value of thermodynamic entropy or increasing value of information entropy is now evidenced locally with the higher growth of the thermodynamic entropy in the environment. The growth of cell structure is now measured by the lengthening of their telomeres.

Druh

J_ost - Ostatní články v recenzovaných periodicích

CEP obor

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OECD FORD obor

10602 - Biology (theoretical, mathematical, thermal, cryobiology, biological rhythm), Evolutionary biology

Projekt

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Návaznosti

N - Vyzkumna aktivita podporovana z neverejnych zdroju

Rok uplatnění

2018

Kód důvěrnosti údajů

S - Úplné a pravdivé údaje o projektu nepodléhají ochraně podle zvláštních právních předpisů

Název periodika

The Pharmaceutical and Chemical Journal

ISSN

2349-7092

e-ISSN

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Svazek periodika

5

Číslo periodika v rámci svazku

4

Stát vydavatele periodika

IN - Indická republika

Počet stran výsledku

10

Strana od-do

71-80

Kód UT WoS článku

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EID výsledku v databázi Scopus

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